Decreased properdin activity in acute glomerulonephritis.
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Absorption↗
Biomedical subjects
R J Pickering
Publications and source records attributed to R J Pickering.
Chronic membranoproliferative glomerulonephritis with hypocomplementemia.
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Adolescent↗
Ontogenetic development of the complement system.
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Animal Nutritional Physiological Phenomena↗
Mechanisms of leukocyte infiltration of allografts. I. The separation and early appearance of two components.
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Animals↗
Studies on the mechanism of canine renal allograft rejection.
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Animals↗
Decreased properdin activity in Swiss-type lymphopenic agammaglobulinemia.
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Agammaglobulinemia↗
Significance of abnormalities of serum complement in human disease.
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Complement System Proteins↗
Complement and complement component activities in diseases associated with repeated infections and malignancy.
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Adolescent↗
The complement profile in acute glomerulonephritis systemic lupus erythematosus and hypocomplementemic chronic glomerulonephritis. Contrasts and experimental correlations.
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Animals↗
The complement system in hereditary angioneurotic oedema--a new perspective.
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Agglutination Tests↗
Decreased C'-1q protein concentration and agglutinating activity in agammaglobulinaemia syndromes: an inborn error reflected in the complement system.
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Agammaglobulinemia↗
Complement inactivation by serum from patients with acute and hypocomplementemic chronic glomerulonephritis.
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Adolescent↗
Deficient complement fixation by aggregated gamma globulin from hypogammaglobulinemic patients.
We found that several normal samples of aggregated gamma globulin consistently fixed essentially equal amounts of guinea pig complement, while samples of gamma globulin from three of four hypogammaglobulinemic patients were markedly deficient in this biologic function. Thus, the previously observed limited heterogeneity of gamma globulin in patients with hypogammaglobulinemia is also associated with inefficiency in at least one biological function.
Agammaglobulinemia↗
Complement activity and inflammatory neutrophil exudation in man. Studies in patients with glomerulonephritis, essential hypocomplementemia and agammaglobulinemia.
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Acute Disease↗
Complement-dependent biological functions in complement deficiency in man.
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Antigen-Antibody Reactions↗