Biomedical subjects
R J Dash
Publications and source records attributed to R J Dash.
Relationship between thyrotropin, thyroid hormones, thyroxine-binding globulin and estradiol-17 beta levels in normal pregnancy.
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Pathogenesis and treatment of diabetic ketoacidosis.
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Concurrent handmirror cell variant of acute lymphoblastic leukaemia and infectious mononucleosis.
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Prevalence of abnormal hemoglobins, thalassaemia and erythrocyte glucose-6-phosphate dehydrogenase deficiency in Kashmiris: a preliminary study.
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Bardet Biedl syndrome: a case report with special reference to studies on hypothalamo-pituitary testicular axis.
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Acquired haemoglobin-H like disease.
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Acute effects of histamine H1- and H2-receptor antagonists on pituitary hormone secretion in man.
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Effect of propranolol on serum T3, T4 and TSH, and the TRH induced T3, T4 and TSH responses in euthyroid men.
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Prolactin--I (first of two parts).
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Spectrum of haemolytic anaemias in Punjab, North India.
Between July 1974 and June 1978 the diagnosis of haemolytic anaemia was made in 267 patients. Thalassaemia major was the leading cause (40-50%) every year except in 1977, when a sharp rise in drug induced haemolysis in G-6-PD deficient patients (61.3%) was encountered. This was possible due to an increase in incidence of vivax malaria and the use of anti-malarial drugs in that year. Abnormal haemoglobins were noted in 9.7% of the total number of patients. Hereditary spherocytosis, autoimmune haemolytic anaemias and paroxysmal nocturnal haemoglobinuria formed a minor cause. Others included copper sulphate poisoning, snake bite, hornet sting and Plasmodium falciparum infection.
Idiopathic myelofibrosis without splenomegaly and with an acquired haemoglobin disorder.
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Clinical, hormonal and histologic studies of adrenal cortex in Cushing's disease.
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Haemoglobin E--thalassaemia in Punjab.
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LH, FSH responses to GnRH in lepromatous leprosy.
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Effect of chlorpromazine on human growth hormone.
The authors studied basal HGH levels and HGH response to insulin-induced hypoglycemia in 12 schizophrenic patients who had been treated with 200--450 mg/day of chlorpromazine for 6 months to 4 years compared with 12 schizophrenic patients who had received no drugs and 15 normal control subjects. They found no significant differences among the three groups in basal HGH levels or in maximum response HGH levels. No significant correlation was found between duration or dose of chlorpromazine therapy and HGH secretion. Longitudinal study in 5 previously untreated schizophrenic patients during 13 weeks of chlorpromazine administration showed a nonsignificant reduction in HGH response. Thus, the authors' findings fail to demonstrate any significant effect of chlorpromazine on growth hormone secretion in man.
Age related changes in gonadotropin receptors in testes of rats.
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Leydig cell function in relation to circulating testosterone & LH in rats during postnatal development.
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