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Biomedical subjects

P Jallon

Publications and source records attributed to P Jallon.

68 records · Page 4Linked to original sources
PubMed1987

[Descriptive epidemiology of epilepsy].

More than 100 surveys in more than 30 countries have reported the incidence rate and/or prevalence rate for epilepsy. They have yielded conflicting figures, as well for crude as for specific rates. Some reasons for these discrepancies are analysed: lack of agreement on terminology concerning either epileptic seizures or epileptic syndromes, inclusion or exclusion of certain categories of patients and differences in sources of ascertainment, each of them exposing to some bias. In the state of the art, not a single study can be generalized to the entire population. However, almost all of them may be used with a cautious interpretation of results.

Age Factors↗
PubMed1983

[Severe visceral necrotizing angiitis with hereditary immune disorders: a case].

The authors report a new case of necrosing angeitis similar to those individualised by Churg and Strauss in 1951. After a persistent rhinitis, the patient developed serious asthma, left hemiplegia 8 months later (secondary to an intracerebral hemorrhage) polyneuritis and extensive necrosis of the small intestine. There was no renal or cardiac involvement. Anatomopathological examination of the small intestine revealed an arteriolitis with periarterial granuloma and severe inflammation. Complementary investigations revealed a hypereosinophilia, an inflammatory syndrome, a reduction in the hemolytic 50 complement and of the C4 fraction. Corticosteroid therapy associated with cyclophosphamide formed the basis of treatment. Plasmapheresis have been a useful aid. The follow-up is too short to assess the long term prognosis. A familial immune deficiency, disseminated lupus erythematosis in a sister, and rheumatoid purpura in one of the patient's children are interesting features of this text.

Adult↗
PubMed1981

[Epilepsies of the young adult (author's transl)].

Data on 614 epileptic patients whose first seizure occurred between the ages of 19 and 29 were reviewed and some clinical and electroencephalographical features compared, in order to clear up the semiology and etiology of this type of late-onset epilepsy. Partial epilepsies were more numerous than generalized epilepsies and post-traumatic epilepsies the more frequent. We could only study the evolution of 305 patients for 5 years or more: 60% were not stabilized but, probably, those who were free of seizures no longer consulted the physician. It seems difficult to compare our findings with other studies, the age periods in the latter being too broad (childhood epilepsy, late or very late onset epilepsy).

Adult↗
PubMed1977

[Transverse axial computerised tomography of the brain in partial epilepsy].

Two hundred epileptics presenting with partial epileptic attacks were subjected to investigation by Acta-Scanner. Results were normal in 100 cases. Also found were 20 tumours (13 of them astrocytomas), 7 epileptogenic softenings, 41 cerebral atrophies, 8 of which were porencephalies, 5 calcifications (3 isolated, 2 multiple, connected with Bourneville's disease), 5 post-operative lesions and 22 dubious conditions. Various correlations between the results of scanning, the clinical findings and the E.E.G. are considered. The value of scanning in screening for tumours developing under the cloak of isolated epilepsy is emphasized and attention is drawn to certain difficulties of interpretation.

Adolescent↗
PubMed1976

[Iatrogenic myoclonic encephalopathies caused by bismuth salts].

Since the beginning of 1974, we have studied 17 cases of iatrogenic encephalopathy due to the ingestion of bismuth taken for the treatment of chronic digestive disorders. More than 100 similar cases have been reported in France within the same period. The clinical picture is remarkably consistent: there is a relatively long prodromal phase with difficulties in gait and writing, followed by a phase characterised by four signs, namely a severe confusional syndrome, myoclonus, astasia-abasia and sisorders of language. In our cases, after withdrawal of bismuth, recovery invariably began within 2 to 3 weeks but fatal cases have been described. All our patients had taken, for periods of between 3 weeks and 20 years, bismuth subnitrate. The levels of bismuth in the blood and urine in these patients were between 10 and 100 times as great as those in patients who had taken the same treatment without ill effect. The exact mechanism by which bismuth causes this complication, described only recently, is totally unknown.

Adult↗