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O Hue

Publications and source records attributed to O Hue.

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The effect of multi-cycle-run blocks on pulmonary function in triathletes.

BACKGROUND: This study was designed to determined the pulmonary responses elicited by multi-cycle-run exercise in triathletes and to compare them to those elicited during a simple cycle-run succession. METHODS: Twelve male triathletes underwent three successive laboratory trials: 1) an incremental cycle test, 2) 30 min of cycling followed by 20 min of running (C-R), and 3) five repeated bouts of 6 min of cycling and 4 min of running (X-CR). Before and 10 minutes after the third and fourth trials, the triathletes underwent lung function testing, especially spirometry and diffusing capacity testing for carbon monoxide (DL(CO)). During all trials, ventilatory data were collected every minute using an automated breath-by-breath system. RESULTS: The results showed that: 1) the cardiorespiratory responses observed during running were greater in the X-CR trial for VE/VCO2 and HR, 2) DL(CO) and DL(CO)/VA were significantly reduced after both trials, and 3) there were no significant changes in pulmonary volumes. CONCLUSIONS: We concluded that 1) the multi-block trial elicited greater cardioventilatory responses than simple the cycle-run succession and 2) multi-block seems a good method to stimulate the specific adaptations required for the cycle-run succession, and particularly for the cycle-run transition. In any case, the efficacy of the multi-block model needs to be more thoroughly evaluated over the course of a longer-term training programme.

Adult↗

DLCO response to experimental cycle-run succession in triathletes.

BACKGROUND: We still know relatively little about the factors that define the ability to perform a good run after cycling in triathlon, however, and the perception of discomfort during the first minutes of this post-cycling running has yet to be satisfactorily explained. The pulmonary diffusion capacity for carbon monoxide (DLCO) has been demonstrated to be impaired after the cycle-run succession. Numerous causes have been suggested to explain this phenomenon, but the exact mechanism has not yet been determined. METHODS: Thirteen young male triathletes participated in four different exercise trials: 30 min of cycling followed by 20 min of running (C-R, 1 min rest between C and R), 30 min of running followed by 20 min of running (R-R, 1 min rest between R and R), 30 min of cycling (C), and 30 min of running (R). DLCO and alveolar volume were simultaneously measured during 9 sec of breath-holding before and 10 min after exercise. The transfer coefficient (KCO=DLCO/VA) was then calculated. During all trials, ventilatory data were collected every minute using an automated breath-by-breath system. RESULTS: The results showed that 1) C-R and C induced significant and identical decreases in DLCO and KCO in post-trial compared with pre-trial measurement (40.41+/-2.24 vs 43.49+/-2.36 ml x min(-1) x mm Hg(-1), p<0.01, and 39.37+/-2.16 vs 42.99+/-2.38 ml x min(-1) x mm Hg(-1), p<0.02, for C-R and C, respectively) and 2) there were no DLCO decreases in post-trial compared with pre-trial measurement in R-R and R. CONCLUSIONS: We concluded that cycling exercise in itself seems to increase the immediate post-exercise DLCO impairment.

Adult↗

The effects of drafting on stroking variations during swimming in elite male triathletes.

The aim of this study was to determine the effects of drafting behind another swimmer on the metabolic response and stroke characteristics. Six highly trained male triathletes performed two maximal 400-m swims, one in a drafting (D) and one in a non-drafting condition (ND). Their metabolic response was assessed by measuring the oxygen uptake (VO2) and the blood lactate concentration at the end of each 400 m. Swimming velocity, stroke frequency, stroke length, and stroke index (velocity multiplied by stroke length) were recorded every 50 m. In the D and ND conditions, there was no difference in VO2 [66.7 (1.7) ml x kg(-1) x min(-1) vs 65.6 (1.2) ml x kg(-1) min(-1) respectively], however, the lactate concentrations were lower in D than in ND [9.6 (0.9) mM vs 10.8 (0.9) mM, respectively, P < 0.01]. In D, the performance [1.39 (0.02) m x s(-1) vs 1.34 (0.02) m x s(-1), respectively, P < 0.01] and the stroking parameters (i.e., stroke length and stroke index) increased significantly, while the stroke frequency remain unchanged. In D, a stable pace was maintained, while in ND, velocity decreased significantly throughout the 400 m. In D, the performance gains were related to the 400-m D velocity (r = 0.78, P < 0.05), and to the body fat mass (BFM, r = 0.99, P < 0.01). The stroke index in D was also related to BFM (r = 0.78, P < 0.05). Faster and leaner swimmers achieved greater performance gains and stroke index when drafting. Thus, drafting during swimming increases the performance and contributes to the maintenance of stable stroking parameters such as stroke frequency and stroke length during a 400-m swim.

Adult↗

Performance level and cardiopulmonary responses during a cycle-run trial.

To determine the effect of triathlete performance level on the cardiorespiratory responses elicited by the cycle-run succession, eight regionally and nationally-ranked (Competitive) and five internationally-ranked (Elite) male triathletes underwent four successive laboratory trials: 1) an incremental treadmill test, 2) an incremental cycle test, 3) 30 min of cycling followed by 20 min of running (C-R), and 4) a 20-min control run (R) at the same speed as the run in C-R. Before and 10 min after the third and fourth trials the triathletes underwent lung function testing: spirometry and diffusing capacity testing for carbon monoxide (DL(CO)). During the C-R trial blood samples were drawn to measure venous lactate concentration. During all trials ventilatory data were collected every minute using an automated breath-by-breath system. The results showed that 1) the oxygen uptake (VO2) of post-cycling running versus running alone was similar for both groups; 2) the ventilatory responses (VE, VE/VO2, VE/VC02 and f) of C-R running versus R were significantly higher (P < 0.005) for the Competitive group; and 3) a significant decrease (P< 0.05) in DL(CO) was also noted after the C-R trial in the Competitive group but not in the Elite group. We concluded that 1) the ventilatory responses during a run subsequent to cycling may be related to the triathlete performance level, and 2) the C-R trial induced specific alterations in pulmonary function that may be associated with respiratory muscle alteration and exercise-induced hypoxemia in the Competitive triathletes.

Adult↗

Catecholamine, blood lactate and ventilatory responses to multi-cycle-run blocks.

PURPOSE AND METHODS: This study was designed to determine whether the physiological responses elicited during the run part of repeated bouts of cycle-run exercise are similar to those required during the run segment of a cycle-run succession. Thirteen male triathletes underwent four successive laboratory trials: 1) an incremental treadmill test, 2) an incremental cycle test, 3) 30 min of cycling followed by 20 min of running (C-R), and 4) five repeated bouts of 6 min of cycling and 4 min of running (X-CR). During the C-R and X-CR trials, venous blood samples were obtained to measure lactate, epinephrine and norepinephrine concentrations. During all trials, ventilatory data were collected every min using an automated breath-by-breath system. RESULTS: The results showed that 1) the cardiorespiratory responses observed during running were similar in the X-CR and C-R trials, 2) the lactate concentration was similar in both trials, 3) the epinephrine concentration was greater (277.9 +/- 11.9 vs 169.8 +/- 86.7 pg x mL(-1), P < 0.025) in X-CR than in C-R, and 4) the norepinephrine concentration was similar in both trials, except at the first cycle-run succession (T1) of X-CR. CONCLUSION: We concluded that 1) multi-block training is a good method to stimulate the specific adaptations required for the cycle-run succession, and particularly for the cycle-run transition, and 2) multi-block training seems to induce a greater catecholaminergic response, which may be due to a combination of an inherent effect of this type of training and the triathletes' relative lack of experience with it. In any case, the efficacy of the multi-block model needs to be more thoroughly evaluated over the course of a longer-term training program.

Adult↗

Ventilatory threshold and maximal oxygen uptake in present triathletes.

The aim of this study was to determine the physiological profile of young triathletes who began triathlon competition as their first sport. Twenty-nine male competitive triathletes (23 regionally and nationally ranked triathletes and 6 elite, internationally ranked triathletes) performed two tests, one on a cycle ergometer (CE VO2max) and one on a treadmill (TM VO2max). Results showed (a) no difference between CE VO2max and TM VO2max in the triathletes (69.1 +/- 7.2 vs. 70.2 +/- 6.2 mL x kg(-1) x min(-1), respectively), (b) values of CE VO2max and TM VO2max in elite triathletes (75.9 +/- 5.2 and 78.5 +/- 3.6 mL x kg(-1) x min(-1), respectively) that were comparable to those reported in elite single-sport athletes in these specialities, and (c) although the ventilatory threshold (Th(vent)) was similar in CE and TM, TM Th(vent) was consistently lower for triathletes than TM Th(vent) usually reported for runners.

Adaptation, Physiological↗

Metabolic and cardioventilatory responses during a graded exercise test before and 24 h after a triathlon.

Previous studies have reported respiratory, cardiac and muscle changes at rest in triathletes 24 h after completion of the event. To examine the effects of these changes on metabolic and cardioventilatory variables during exercise, eight male triathletes of mean age 21.1 (SD 2.5) years (range 17-26 years) performed an incremental cycle exercise test (IET) before (pre) and the day after (post) an official classic triathlon (1.5-km swimming, 40-km cycling and 10-km running). The IET was performed using an electromagnetic cycle ergometer. Ventilatory data were collected every minute using a breath-by-breath automated system and included minute ventilation (V(E)), oxygen uptake (VO2), carbon dioxide production (VCO2), respiratory exchange ratio, ventilatory equivalent for oxygen (V(E)/VO2) and for carbon dioxide (V(E)/VCO2), breathing frequency and tidal volume. Heart rate (HR) was monitored using an electrocardiogram. The oxygen pulse was calculated as VO2/HR. Arterialized blood was collected every 2 min throughout IET and the recovery period, and lactate concentration was measured using an enzymatic method. Maximal oxygen uptake (VO2max) was determined using conventional criteria. Ventilatory threshold (VT) was determined using the V-slope method formulated earlier. Cardioventilatory variables were studied during the test, at the point when the subject felt exhausted and during recovery. Results indicated no significant differences (P > 0.05) in VO2max [62.6 (SD 5.9) vs 64.6 (SD 4.8) ml x kg(-1) x min(-1)], VT [2368 (SD 258) vs 2477 (SD 352) ml x min(-1)] and time courses of VO2 between the pre- versus post-triathlon sessions. In contrast, the time courses of HR and blood lactate concentration reached significantly higher values (P < 0.05) in the pre-triathlon session. We concluded that these triathletes when tested 24 h after a classic triathlon displayed their pre-event aerobic exercise capacity, bud did not recover pretriathlon time courses in HR or blood lactate concentration.

Adolescent↗

Ventilatory responses during experimental cycle-run transition in triathletes.

PURPOSE AND METHODS: To determine the effects of cycling on a subsequent triathlon run, nine male triathletes underwent four successive laboratory trials: 1) an incremental treadmill test, 2) an incremental cycle test, 3) 30 min of cycling followed by 5 km of running (C-R), and 4) 30 min of running followed by 5 km of running (R-R). Before and 10 min after the third and fourth trials, the triathletes underwent pulmonary function testing including spirometry and diffusing capacity testing for carbon monoxide (DL(CO)). During the C-R and R-R trials, arterialized blood samples were obtained to measure arterial oxygen pressure (PaO2). During all trials, ventilatory data were collected every minute using an automated breath-by-breath system. RESULTS: The results showed that 1) the oxygen uptake (VO2) observed during subsequent running was similar for the C-R and R-R trials; 2) the ventilatory response (VE) during the first 8 min of subsequent running was significantly greater in the C-R than in R-R trial (P < 0.05); 3) only the C-R trial induced a significant increase (P < 0.05) in residual volume (RV), functional residual capacity (FRC), and the ratio of residual volume to total lung capacity (RV/TLC); and 4) although a significant decrease (P < 0.05) in DL(CO) was noted after C-R, no difference between the two exercise trials was found for the maximal drop in PaO2. CONCLUSIONS: We concluded that 1) the C-R trial induced specific alterations in pulmonary function that may be associated with respiratory muscle fatigue and/or exercise-induced hypoxemia, and 2) the greater VE observed during the first minute of running after cycling was due to the specificity of cycling. This reinforces the necessity for triathletes to practice multi-trial training to stimulate the physiological responses experienced during the swim-cycle and the cycle-run transitions.

Adolescent↗

The influence of prior cycling on biomechanical and cardiorespiratory response profiles during running in triathletes.

The aim of the present study was to determine the effects of 40 km of cycling on the biomechanical and cardiorespiratory responses measured during the running segment of a classic triathlon, with particular emphasis on the time course of these responses. Seven male triathletes underwent four successive laboratory trials: (1) 40 km of cycling followed by a 10-km triathlon run (TR), (2) a 10-km control run (CR) at the same speed as TR, (3) an incremental treadmill test, and (4) an incremental cycle test. The following ventilatory data were collected every minute using an automated breath-by-breath system: pulmonary ventilation VE, l x min[-1]), oxygen uptake (VO2, ml x min(-1) x kg[-1]), carbon dioxide output (ml x min[-1]), respiratory equivalents for oxygen (VE/VO2) and carbon dioxide (VE/VCO2), respiratory exchange ratio (R) respiratory frequency (f, breaths x min[-1]), and tidal volume (ml). Heart rate (HR, beats x min[-1]) was monitored using a telemetric system. Biomechanical variables included stride length (SL) and stride frequency (SF) recorded on a video tape. The results showed that the following variables were significantly higher (analysis of variance, P < 0.05) for TR than for CR: VO2 [51.7 (3.4) vs 48.3 (3.9) ml x kg(-1) x min(-1), respectively], VE [100.4 (1.4) l x min(-1) vs 84.4 (7.0) l x min(-1)], VE/VO2 [24.2 (2.6) vs 21.5 (2.7)] VE/VCO2 [25.2 (2.6) vs 22.4 (2.6)], f[55.8 (11.6) vs 49.0 (12.4) breaths x min(-1)] and HR [175 (7) vs 168 (9) beats x min(-1)]. Moreover, the time needed to reach steady-state was shorter for HR and VO2 (1 min and 2 min, respectively) and longer for VE (7 min). In contrast, the biomechanical parameters, i.e. SL and SF, remained unchanged throughout TR versus CR. We conclude that the first minutes of the run segment after cycling in an experimental triathlon were specific in terms of VO2 and cardiorespiratory variables, and nonspecific in terms of biomechanical variables.

Adult↗

Blood rheological responses to running and cycling: a potential effect on the arterial hypoxemia of highly trained athletes?

To investigate 1) the blood rheological responses to high training volume and 2) the potential effect of these responses on arterial hypoxemia induced during submaximal running and cycling, 10 triathletes performed an incremental cycle test, 20 minutes of running (R), and 20 minutes of cycling (C). All trials were performed at nearly 75 % of VO2max. Hematocrit (H), blood viscosity (etab), plasma viscosity (etapl), index of erythrocyte rigidity (Tk), changes in plasma volume (DeltaPV), pulmonary diffusing capacity (DLco), and arteriolized blood gas (PaO2) were measured before and after each trial. Pulse oxymetry (SpO2) and cardioventilatory data were collected continuously. A significant increase in etab, etapl, and H was noted after R and C with respect to pre-exercise, whereas DeltaPV decreased, with a greater decrease after C. Tk was significantly higher after R than after C. A significantly greater drop in DLco was noted after C compared with R. SpO2 decreased significantly more during R, as did PaO2. We conclude that blood rheological responses are specific to running and cycling. Cycling induced a sharp decrease in plasma volume, which could partially explain the greater DLco alteration. Running was characterized by an increase in Tk, which could be implicated in the severity of the drop in arterial oxygenation observed.

Adult↗

[Occlusal anatomy].

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Deglutition↗