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Biomedical subjects

N Yoshida

Publications and source records attributed to N Yoshida.

At least 667 records · Page 37Linked to original sources
PubMed1977

Responses to the postural change and orthostatic dysregulation symptoms: a population study on Japanese junior and senior high school students.

Unselected 580 male and 779 female junior and senior high school students were subjected to the tilting test and informations on 11 autonomic dysregulation symptoms and complaints were taken by interviews. Results of the analyses of the data are summarized as follows: 1) Number of symptoms and complaints possessed by each individual distributes continuously but nonrandomly among testees; that is, individuals having no symptom and those having 5 or more symptoms are significantly more numerous than those expected from random distribution, indicating accumulation of the symptoms into some individuals. 2) Some specific symptoms tend to coincide with each other. 3) Principal component analyses disclosed that the first principal component can explain only 16-20% of the observed variation in the presence or absence of symptoms and complaints, suggesting that numerous factors are responsible for the occurrence of the symptoms. 4) Measurements of the blood pressures and the pulse rate, and their responses to the postural change show continuous, normal distributions, and significantly correlate to each other. 5) The responses to the tilting test are increased with the increase of number of symptoms and complaints possessed by the testees.

Adolescent↗
PubMed1977

Potentiation by collagen or epinephrine of platelet responsiveness to aggregation. The possible role of substance(s) released from platelet membranes.

When human platelets in plasma were exposed to a small amount (nonaggregating concentration) of collagen, epinephrine, or arachidonic acid, their responsiveness to aggregating agents was potentiated and they were aggregated by a subsequent addition of nonaggregating concentrations of the stimulants. Otherwise the nonaggregating concentrations of the stimulants were incapable of inducing platelet aggregation. The potentiation of platelet responsiveness to aggregating agents was also caused by collagen- or epinephrine-treated platelet membranes. Furthermore, the soluble fraction of collagen- or epinephrine-treated membranes contained some material responsible for platelet potentiation, indicating that the responsible material was released from platelet membranes by collagen or epinephrine. It is suggested that the material may be arachidonic acid or its derivatives related to prostaglandins, since the soluble fraction of collagen- or epinephrine-treated membranes contained a larger amount of the precursor(s) of prostaglandin Falpha than the untreated membranes.

Arachidonic Acids↗