[Electrodestruction of the atrioventricular junction in a patient with post-infarction ventricular tachycardia].
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Biomedical subjects
Publications and source records attributed to N A Cherkasova.
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Central hemodynamic parameters under the effect of mildronate were examined in 62 patients suffering from coronary heart disease, 35 of these with acute myocardial infarction complicated by acute left-ventricular insufficiency and 20 with atherosclerotic cardiosclerosis with chronic cardiac insufficiency. The drug effect was assessed in two groups of patients after a single intravenous injection of 0.5-3 g. In group 1 (n = 53) mildronate effect on central hemodynamic parameters was assessed in spontaneous cardiac rhythm. Variously directed statistically unreliable hemodynamic shifts were revealed, related to heart rhythm changes. In group 2 (n = 9) the drug effect on heart rhythm was eliminated with the use of two-chamber electrocardiostimulation, and various hemodynamic regimens with hypo-, eu-, and hyperkinetic circulation types were artificially created by changing the A-V interval. Mildronate had no effect on the hemodynamics during two-chamber electrocardiostimulation.
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Cardiac output was investigated in coronary patients with temporary (15) and permanent (16) programmed two-chamber electrostimulation of the heart, using different induced atrioventricular interval values. The study was based on integral rheoplethysmography, peripheral volumetric sphygmography, sectoral echocardiography and two-dimensional doppler echocardiography with color code. The adjustment of atrioventricular interval in two-chamber cardiac electrostimulation can modulate cardiac output by means of changing the duration and rate of left-ventricular filling flow, produced by the atrial systole.
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Explore the source record for details and available documents.
Explore the source record for details and available documents.