Biomedical subjects
M Wanic-Kossowska
Publications and source records attributed to M Wanic-Kossowska.
[Ventilation and gas exchange in patients with chronic renal failure treated with hemodialysis (HD) and intermittent peritoneal dialysis (IPD)].
In 18 patients with chronic renal failure treated by hemodialysis (HD) before, during and after dialysis procedure breathing patterns and blood gases were estimated. Significant changes in PdCO2 and PvCO2 during HD may confirm hypothesis that CO2 diffusion into the dialysis fluid play a role in hypoventilation and hypoxemia during HD. PaO2 decrease in patients treated by peritoneal dialysis (PD) after infusion 21 of fluid into the peritoneal cavity. Increase in minute ventilation (VE) and oxygen consumption (ViO2) at 2nd and 4rs hour of PD indicate that substrate metabolism during dialysis relates to alternations in ventilation. In all patients before dialysis treatment presence of ventilation disturbances of restrictive typ were demonstrated with decreased vital capacity (VC), reduced maximal ventilation (MBC) and lower one-second forced expiratory volume (FEV1). Residual volume (RV) was significantly higher. After HD we observed a significant increase of total lung capacity (TLC) and decrease of RV, whereas after PD a significant decrease of RV.
[Pulmonary infarction as a complication of thrombotic changes in an arteriovenous fistula of a patient with terminal renal insufficiency treated with long-term hemodialysis and recombinant human erythropoietin].
The case of 48 year old female patient with terminal renal insufficiency treated with hemodialysis and human recombinant erythropoietin was observed because of pulmonary infraction. The symptoms of disease were observed in the right lung and in region of arteriovenous fistula. It was documented that therapy with human recombinant erythropoietin may induce thrombosed complications during hemodialysis with arteriovenous fistula.
[Echocardiographic evaluation of left ventricular function in patients with chronic renal failure treated with intermittent peritoneal dialysis].
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[Aluminum toxicity in patients with chronic renal failure].
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[Effect of aminophylline on respiratory function in patients with chronic renal failure treated by peritoneal dialysis].
In 8 patients with chronic renal failure aminophylline influence was studied on respiratory function. No changes were seen in lung volumes after 30 minutes of intravenous aminophylline infusion. After 2 weeks of intraperitoneal infusion of aminophylline, maximal ventilation, vital capacity, forced vital capacity and inspiratory residual volume rose significantly. Residual volume and the ratio residual volume/total lung capacity, decreased. This changes may indicate an improved contractility of the respiratory muscles. A rise in the concentration of oxypurines after peritoneal dialysis and a significant improvement in the arterial oxygen tension indicate that aminophylline influences the respiratory function by bronchodilatation and by contractility improvement of the respiratory muscles.
[Effect of carbon dioxide (CO2) pressure on minute ventilation, parameters of gas exchange and blood gases during hemodialysis using fluid containing acetate and bicarbonate buffers].
In 20 patients with chronic renal failure on a hemodialysis with acetate-containing dialysing fluid gasometric, ventilation and breathing patterns disturbances were determined. The loss of CO2 in the dialysate is attributed the major cause of hypoxemia due to alveolar hypoventilation. Hemodialysis with bicarbonate-containing dialysate can be performed in the absence of any change in ventilation and PaO2 despite a systemic alkalosis. Hyperventilation during HD with high concentration of bicarbonate indicate that changes in CO2 tension in the pulmonary circulation can lead to a change in minute ventilation due to the presence of slowly adapting pulmonary chemoreceptors. In patients with low respiratory response, respiratory muscle weakness intensified additionally by hypercapnia may explain this phenomenon.
[Analysis of the causes of respiratory muscle hypotonia in patients with chronic renal failure treated by peritoneal dialysis and hemodialysis].
In 21 patients with chronic renal failure spirometric and electromyographic examinations (including m. pectoralis, m. rectus abdomini, and m. obligus abdomini) were performed before and after hemodialysis. In 10 patients treated by peritoneal dialysis and, in 35 patients treated by hemodialysis serum PTH, thyroid hormones (T4, T3, rT3), CPK, aldolaze, pyruric acid, lactic acid, alkaline phosphatase were determined. In both groups before and after dialysis serum sodium, potassium, calcium, magnesium were determined. We observed negative correlation between PTH and respiratory muscles weakness indices. This fact may confirm the contribution of PTH in uremic myopathy evaluation.
[Respiratory muscle hypotonia as one of the causes of hypoventilation and hypoxemia in patients with chronic renal failure treated by hemodialysis].
In 21 patients with chronic renal failure spirometric and electromyographic (included m. pectoralis, m. rectus abdomini, m. obligus abdomini) examinations were performed before and after hemodialysis. We observed a decreased myoelectrical activity of muscles on the basis of amplitude of EMG. Weakness of the respiratory muscles may be one of the causes of hypoventilation and hypoxemia during hemodialysis.
[Effect of peritoneal dialysis and hemodialysis on respiratory function in patients with chronic renal failure].
Respiratory function was studied in 17 patients with chronic renal failure treated by peritoneal dialysis and in 18 patients treated by hemodialysis. During peritoneal dialysis and hemodialysis we observed impaired respiratory function and changes in lung volumes. Decrease of the arterial oxygen tension during peritoneal dialysis may be due to the intraabdominal fluid volume, in patients treated by hemodialysis--to respiratory muscle weakness.
[Reversible form of hypertension--the page kidney].
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[Prevention of dialysis-induced leukopenia and hypoxemia by using low-molecular dextran].
In 10 patients with end stage renal failure on chronic haemodialysis the effect was studied of the low molecular dextran (filling the dialyser) on the peripheral blood leucocyte count and some gasometric and ventilatory parameters during and after haemodialysis (HD). When the dialyser was filled with dextran neither the leucocyte count nor the arterial PaO2 were altered but on the 10th minute of HD without dextran both those values fell. The decrease of the ventilation rate (VE), of the alveolar oxygen tension (PAO2) as well as of the index of oxygen utilisation (VIO2/VE) within the first hour of HD with or without dextran might be due to the absorption of the CO2 by dialysing fluid.
[2 cases of kidney damage during the treatment with gold preparations].
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[Effect of peritoneal dialysis on respiratory function in patients with chronic renal failure].
In 17 patients with end stage renal failure the effect was studied of a single peritoneal dialysis on the respiratory function, in the sitting and the recumbent position of the body. After the infusion of 2 liters of the dialysis fluid into the peritoneal cavity arterial PaO2 fell as well as the functional residual capacity, but the alveolar/arterial oxygenation difference (A-alpha)O2 rose. The most marked changes accompanied by dyspnea occurred in most patients in the sitting position. The inspiratory residual volume (IRV) increase reflects a better diaphragmatic function. The usefulness of aminophylline is discussed which not only dilates the bronchi but also improves the contractibility of the diaphragmatic muscle.
[Sclerosing peritonitis in a patient treated with intermittent peritoneal dialysis].
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[Disorders of the respiratory function in patients with chronic renal failure treated by hemodialysis. I. Evaluation of the functional state of the respiratory system].
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[Disorders of the respiratory function in patients with chronic renal failure treated by hemodialysis. II. Evaluation of pulmonary gas exchange and hemodialysis-related leukopenia].
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