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Biomedical subjects

M Mitomo

Publications and source records attributed to M Mitomo.

At least 55 records · Page 3Linked to original sources
PubMed1986

Radiation necrosis of the brain and radiation-induced cerebrovasculopathy.

Thirteen patients with radiation necrosis and 6 patients with occlusive radiation-induced cerebrovasculopathy were reviewed. Computed tomography (CT) demonstrated low attenuation with a mass effect and irregular contrast enhancement in the radiation necrosis. The mass effect and the contrast enhancement decreased gradually. Angiography showed an avascular mass, generalized narrowing of the ipsilateral arteries and slow blood circulation in 3 cases. Radiation-induced vasculopathy appeared as localized stenosis and irregular walls of the arteries within the irradiated area. In cases with radiation vasculopathy accompanied by sufficient collateral blood flow brain damage was rarely demonstrated by CT. In patients with radiation-induced vasculopathy the lesion was not associated with signs of radiation necrosis.

Adult↗
PubMed1985

Focal luxury perfusion with an early-filling vein in relation to neurological symptoms evoked by heat.

A 50-year-old man with a 1-year history of transient attacks of left total hemiparesis was admitted to hospital with a complaint of increasing frequency of attacks. Minimal or moderate left hemiparesis was elicited by elevation of environmental temperature when taking a hot bath or a hot shower. Right carotid angiography revealed an early-filling vein near the right central sulcus. An increase of focal luxury perfusion by elevation of body temperature seemed to cause relative ischaemia in this paracentral gyrus.

Cerebrovascular Circulation↗
PubMed1983

[Angiographic findings of atrophic cerebral disorders in children].

Angiographic findings of the cerebral atrophy in children are discussed. Cerebral angiographies were performed on 20 patients, ranging from 6 months to 16 years of age. Their clinical diagnoses were H-H-E syndrome (4 cases), sequelae of meningoencephalitis (2), sequelae of head trauma (3), infantile spasms (2) and others (9). Final radiological diagnoses of these cases were hemiatrophy (14 cases), generalized atrophy (2) and focal atrophy (4). Circulation time, caliber of arteries, degree of opacification in cortical and deep veins and early filling of deep veins were investigated on angiogram. Slow circulation of cerebral blood flow was shown in 10 cases and normal circulation in 10 cases. Arterial narrowing was observed more frequently in slow circulation group. In cases with hemiatrophy, small caliber of the middle cerebral arteries and slow circulation on the atrophic hemisphere were demonstrated, but no abnormality on the normal side. Deep veins were prominently opacified in cases which showed poor cortical veins, the other way cortical veins were prominent in cases which showed poor visualization of deep veins. Cortical veins and deep veins may compensate each other in cerebral venous drainage. In most cases of slow circulation, dominant opacification of deep veins were visualized and these veins already appeared in arterial phase, but cortical veins had a tendency of poor opacification. These venous drainage patterns are similar to those of Sturge-Weber syndrome. Causes of these vascular changes are unknown. But they may play an important role in cerebral atrophic disorders.(ABSTRACT TRUNCATED AT 250 WORDS)

Adolescent↗