Biomedical subjects
Publications and source records attributed to M Madan.
The authors stated the correlation between the levels of homocarnosine in the amniotic fluid, fetal encephalopathy and the degree of fetal neurologic symptomatology.
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Some sporogenous yeasts (Brettanomyces bruxellensis, Debaryomyces hansenii, Hansenula ciferrii, Hansenula polymorpha, Pichia polymorpha, Saccharomycopsis guttulata, and Saccharomyces chevalieri), isolated from various fruits have been examined for their organic growth factor requisites. H. ciferrii was completely deficient in thiamine, biotin, inositol, riboflavin, niacin, and partially deficient in pantothenic acid. It required an external supply of 0.1-1.0 ppm thiamine, 0.01-0.1 ppm biotin, 10.0 ppm inositol, 0.10 ppm niacin and riboflavin for its optimum growth. H. polymorpha showed partial deficiency only in xanthine. P. polymorpha gave indications of partial deficiencies in thiamine and biotin. S. guttulata was completely deficient in biotin, and partially deficient in adenine sulphate. It required 0.01 ppm biotin for optimum growth. S chevalieri was completely deficient in pyridoxine and partially deficient in thiamine. It required 0.1 ppm pyridoxine for maximum growth. D. hansenii and B bruxellensis were auxoautotrophic for the various growth factors studied.
Trace element studies were carried out on four species of Fusarium: F. moniliforme, F. solani, F. poae and F. bulbigenum. Out of fifteen trace elements tested, Fe, Zn, Mn, Cu, Mo and B were found to be essential for growth and sporulation of all these species of Fusarium. Optimum concentrations in ppm of essential trace elements of these fungi were as follows: F. moniliforme Fe 1.0, Zn 0.1, Mn 0.1, Cu 0.1, Mo 0.1, B 1.0-10.0; F. solani Fe 1.0, Zn 10.0, Mn 0.1, Cu 0.01, Mo 1.0, B 0.1; F. poae Fe 1.0, Zn 0.1, Mn 0.1, Cu 1.0, Mo 0.1, B0.1, F. bulbigneum Fe 10.0, Zn 1.0, Mn 1.0, Cu 1.0, Mo 0.01, B 1.0. Concentrations higher than the optimum were inhibitory to the respective fungi.
Penicillium expansum Link showed excellent/good growth on all carbohydrates except ribose, lactose, sorbose and sucrose, which induced fair or poor growth. It made excellent growth on mannitol and dulcitol while the rest of the carbon sources were either poorly utilized or not utilized. Sorbose neither inhibited nor promoted growth of the pathogen in the presence of good carbon sources. The pathogen showed fair/poor growth on cystine, phenylalanine, histidine, mono-HCl, histidine di-HCl, ammonium sulphate, ammonium nitrate, ammonium chloride, ammonium phosphate, and urea, while the rest of the nitrogen sources induced excellent/good growth. However, there was no growth on potassium nitrite. Cystine inhibited the growth of the pathogen to some extent in the presence of good amino acids, i.e. serine proline, methionine, arginine mono-HCl, tyrosine and tryptophane, and not at all were inhibited in the presence of the remaining amino acids.
Plasm progesterone and cortisol were measured in jugular blood by competitive protein-binding assay. Six cycling, estrus-synchronized Guernsey heifers were followed for four consecutive estrous cycles under two controlled temperature (18.2 C, 55% relative humidity; and 33.5 C, 55% relative humidity) conditions. Prolonged exposure to heat of Guernsey heifers increased plasma progesterone on days 2 to 19 of the first cycle and only on days 2 to 8 of the second cycle. However, heat stress depressed plasma cortisol in both cycles and reduced the correlation coefficient between these steroids relative to specific stages of the estrous cycle.
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The natural history of visceral artery aneurysms, and in particular those of the hepatic artery, is unclear. The clinical presentation can include upper abdominal pain, bleeding into the gastrointestinal tract, and obstructive jaundice. We present the case of a middle-aged woman with right upper quadrant pain and a palpable mass, in whom a thrombosed hepatic artery aneurysm was found to be the cause.