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Biomedical subjects

M M Bassan

Publications and source records attributed to M M Bassan.

29 records · Page 2Linked to original sources
PubMed1978

Prosthetic valve hemolysis aggravated by gastrointestinal bleeding.

A case is presented of a patient with 2 prosthetic valves and a mild chronic hemolytic anemia, in whom the hemolysis was suddenly aggravated by the occurrence of an acute gastrointestinal hemorrhage. This increased hemolysis was readily reversed by transfusion. It is postulated that the pathogenetic mechanism of this process was the increased direct crushing trauma and turbulence, both of which resulted from the tachycardia initiated by the gastrointestinal hemorrhage. The importance of avoiding or correcting tachycardia in patients with prosthetic valves is emphasized.

Adult↗
PubMed1977

Pericardial tamponade due to perforation with a permanent endocardial pacing catheter.

A case is presented in which shock developed suddenly during implantation of a permanent pacemaker. The cause proved to be pericardial tamponade secondary to catheter perforation. Analysis of this case and the 6 similar cases reported in the literature reveals that although perforation occurs not infrequently from immediately to many months after implantation, tamponade is quite rare and is invariably closely related temporally to catheter manipulation. Tamponade has occurred with different catheter sizes and makes. It is more likely related to local myocardial factors than to faulty technique. Closed pericardiocentesis is generally unsuccessful in relieving the tamponade, and prompt open drainage through the subxiphoid approach is recommended.

Aged↗
PubMedDate not supplied

Who's afraid of angina pectoris? Separating the symptom from the marker.

100 cardiologists were asked for their views on the risk associated with an episode of exertional angina in patients with stable angina. 58% thought that such an episode carried some risk of permanent damage or death, and 78% advised their patients to avoid anginal pain. In contrast, exercise studies done during the evaluation of anti-anginal drugs indicate that exertional angina can be provoked frequently and repeatedly without apparent risk, and other studies have shown that repeated exercise to the onset of angina is not only safe but improves exercise tolerance. Epidemiological investigations suggest that sudden death and myocardial infarction do not commonly occur during exertion, and our knowledge of pathology indicates that both events are usually caused by acute coronary thrombosis. Many physicians seem to treat the symptom of angina because they are unduly motivated by fear of the underlying potentially fatal disease for which angina is a marker. Such an attitude will tend to cause undue anxiety among patients, will lead to unnecessary restriction of patients' activities, and may result in excessive invasive treatment of mild angina.

Angina Pectoris↗
PubMedDate not supplied

Symptomatic sick sinus syndrome due to guanethidine. Case report.

A patient is described who developed symptomatic sinus bradycardia as low as 20 beats per minute and sinus arrest of up to 4.4 seconds while receiving guanethidine, 75 mg daily. The bradycardia resolved following discontinuation of the drug and reappeared upon challenge with it. Intrinsic disease of the sinoatrial and atrioventricular nodes was evidenced 3 weeks following discontinuation of the guanethidine by a borderline abnormally prolonged sinus node recovery time of 1500 msec and a PR interval of 0.28 seconds. Although sinus bradycardia is a known and not infrequent side effect of guanethidine, such an extreme form as seen in our patient appears to be quite rare, and may be related to the pre-existing disease of the conduction system.

Aged↗
PubMedDate not supplied

Wolff-Parkinson-White syndrome mimicking myocardial infarction: unmasking by means of rapid atrial pacing.

A patient is presented who had Q waves in Leads II, III, and aVF suggestive of inferior myocardial infarction. Despite a normal P-R interval, Wolff-Parkinson-White syndrome was suspected because of an abnormal delta wave-type QRS configuration. By means of rapid atrial pacing the accessory pathway was blocked, and the QRS complex was temporarily normalized. The abnormal Q waves disappeared, removing the suspicion of infarction. Rapid atrial pacing is a simple procedure that is recommended for clarifying the electrocardiographic diagnosis when WPW syndrome is suspected.

Adult↗