Search PubMed⌕ Search

Biomedical subjects

M Giroud

Publications and source records attributed to M Giroud.

At least 199 records · Page 11Linked to original sources
PubMed1986

[Migraine and somnambulism. A survey of 122 migraine patients].

A retrospective study looking for an history of somnambulism in childhood, in a population of 122 patients with migraine, and 110 patients with non-migraine headache has been performed. A very high frequency of somnambulism in patients with migraine was found whereas frequency of somnambulism in patients with non-migraine headache was similar to that of the general population. The probability that a sleepwalking child acquires a migraine is greater when she is a girl. Migraine is then often of the ophthalmic type. Somnambulism and migraine appear at different ages, the former in the late infancy, the latter in childhood and both could be due to a disorder of serotonin metabolism. The authors suggest that somnambulism be considered as an accessory criterion for the diagnosis of migrainous disease.

Adolescent↗
PubMed1986

[Non-traumatic rhinorrhea from a latero-sellar fistula of the temporal fossa adjacent to an empty sella turcica. Computed tomography aspects; surgical treatment].

The authors report a case of non traumatic rhinorrhea due to an osteo-dural fistula communicating between the latero-sellar part of the left middle cranial fossa and the sphenoidal sinus. This fistula, probably congenital, was adjacent to an intrasellar arachnoidocele ("empty" sella turcica), but was not responsible for it. This lesion was demonstrated by CT scan associated with the injection of Metrizamide. The surgical treatment (trans-sphenoidal approach) allowed complete cure of the fistula and its associated symptoms.

Cerebrospinal Fluid Rhinorrhea↗
PubMed1986

[Orthostatic arterial hypotension and giant aneurysm of the basilar trunk].

The authors report two observations of giant aneurysm of basilar artery, where the first neurological signs were preceded or escorted by a postural hypotension. They emphasize the place of rhombencephon in the control of blood pressure and they bring together their cases with those reporting a postural hypotension revealing a lesion of medulla. They insist upon the necessity to look a peripheric and central neurological lesion in front of a postural hypotension.

Adult↗
PubMed1985

[Evaluation of changes in gamma-glutamyltransferase in chronic treatment with antiepileptic agents].

To know the intensity of liver enzyme induction during a treatment with anticonvulsant, the authors have measured gamma GT before and at the 7th, 30th, 60th days after a treatment by one of the 4 major anticonvulsant as phenobarbital, diphenylhydantoin, carbamazepine and sodium valproate. All alcoholic patients, and all the patients having a liver disease have been eliminated. The results show that diphenylhydantoin is the most important inductor of gamma GT with an elevation that can reach 312% of basal level, followed by phenobarbital, when sodium valproate and carbamazepine are the weakest inductors. More, induction by carbamazepine in women is more weak than in man. Age takes a place in intensity of induction with a major induction observed between 30 and 50 years old for phenobarbital, and above 50 years old for sodium valproate. These effects are not dependent of an hepatitis. The knowledge of the upper levels of gamma GT induction by anticonvulsant appear to us usefull for several reasons: carbamazepine and sodium valproate being the weakest inductors, they must be chosen in priority in women under contraceptive treatment. Any abnormal elevation of gamma GT need to look of an alcoholic intoxication, an hepatitis or a liver cancer.

Adult↗
PubMed1985

[A unusual manifestation of multiple sclerosis: sciatica].

The authors recall the possibility of a typical sciatica in multiple sclerosis and describe seven new cases. Their main clinical features are: early onset before any other signs of the disease, hyperalgic, paroxysmal, short, repetitive episodes, absence of mechanical or effort triggers, absence of improvement with rest or anti-inflammatory drugs, well defined course. The intra-medullar origin of these attacks is suggested by the clinical criteria and the aspect of evoked potentials of the lower limbs recorded in 2 of the 7 patients.

Adult↗
PubMed1985

[GABAergic control of hypophyseal gonadotropic function and prolactin. Demonstration by valproic acid].

While there is well-known evidence for dopaminergic control over gonadotrophic and Prolactin functions, the way gamma-aminobutyric acid (Gaba) acts and the routes through which it acts are not so well known. Valproic acid stimulates cerebral Gabaergic tracts. It was therefore used by the authors to study first the basal levels of LH FSH and Prolactin and the way they reacted after two months treatment with this anti-convulsant. The authors noted a decrease of LH-RH stimulated LH and a decrease of the basal levels or Prolactin in TRH stimulated Prolactin. So Gaba can be a neuro-modulator of the hypothalamo-pituitary axis, acting synergically with the dopaminergic tracts to decrease Prolactin secretion but opposing the dopaminergic tracts in the control over LH.

Adult↗
PubMed1984

[Bilateral parietal syndrome approximating a Balint syndrome].

A patient presented with disorders of saccadic and pursuit movements a wandering gaze, the impossibility to maintain fixation, a concentric shortening of visual attention mainly of the right side, an incomplete right homonymous hemianopia (respecting 10 to 20 degrees), and optical and auditory ataxia predominantly affecting the right hand. Pathology showed a large left parietal glioma in the white matter in the superior and inferior parietal lobes, with microscopic evidence of parietal cortex invasion. A hematoma on the right side was situated entirely parasagitally in the precuneus, approaching the cortex at this level. The two lesions were independent and the splenium was intact. The frontal lobes were normal. A review of nearly 30 cases of bilateral syndrome of the parieto-occipital junction showed that anomalies of visually guided ocular movements were always present. These cases require differentiation from those, such as the present case, which involve mainly a visuomotor coordination disorder and from those with predominant visual disorientation.

Ataxia↗