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Biomedical subjects

M F Ismagilov

Publications and source records attributed to M F Ismagilov.

18 recordsLinked to original sources
PubMed1993

[The genetic aspects of neurogenic syncopes].

The authors specified the prevalence of syncopal states in the representative sample of 2309 schoolchildren from the city of Kazan, as well as hereditary factors in the genesis of neurogenic syncopes basing on the clinico-genealogical analysis of 115 families. 91.4% of the syncopal conditions were considered to be neurogenic syncopes (populational frequency is 6.41%) and family disease close by relevant characteristics to multifactorial illnesses with rather high (73.5%) proportion of the genetic factors. When the parents remarried by type patient-to-patient, the segregation rate (0.43) of neurogenic syncopes is close to the expected (0.50) though this does not rule out a dominant model of monogenic affection.

Adult↗
PubMed1992

[HLA antigens in persons with a predisposition to frequent sympathetic-adrenal paroxysms].

Comparison of the rate of HLA antigens I in 81 patients with frequently occurring sympathoadrenal paroxysms (SAP) associated with vegetovascular dystonia and 113 healthy subjects revealed the increase of the number of antigen B12 carriers among the patients as compared to the healthy subjects' group. The number of antigens A9, B35 in SAP patients was elevated whereas that of B18, B38 and B40 decreased. SAP related to age-associated hormonal rearrangements were marked by antigens Cw4 and A1, the grave SAP patterns by B12, SAP without nocturnal paroxysms and the early disease onset by the lack of antigen A19. The dominant gene that determines the onset of SAP is localizes in the HLA area. It may be associated with a hypothetical gene analogous to the locus implicated in the determination of the threshold of neuromuscular excitability of rats. It is not excluded that such a relationship is mediated via the blood content of Mg2+.

Adrenal Gland Diseases↗
PubMed1989

[Psychoautonomic syndrome in non-infectious subfebrile conditions in children].

To ascertain the cerebral organization and the importance of its specific levels in the occurrence of the thermopathological syndrome associated with vegetovascular dystonia, 40 children aged 8-15 years with non-infectious subfebrile conditions of long standing were examined. The children's status was marked by polysystemic shifts common to the neurogenous disorders. Of crucial significance was dysfunction of the structures of the suprasegmental level (the limbic-reticular complex) of vegetative regulation with the involvement of the specialized hypothalamic centers of thermoregulation, with excessive sympathetic influences at the periphery and formation of the psychovegetative syndrome. An individual analysis of the psychovegetative syndrome makes it possible to define the contribution of the concrete endo- and exogenous factors to the pathogenesis of non-infectious subfebrile conditions and to develop a differential approach to the treatment.

Adolescent↗
PubMed1988

[The prognosis of the development of autonomic dysfunction in puberty as the source of psychosomatic diseases in adults].

Comparative study of the incidence of various hereditary, constitutional and environmental factors in two groups of schoolchildren aged 8-18 with (506 subjects) and without (604) clinical signs of autonomic dysregulation yielded a set of indices reliably correlating with the autonomic dysfunction. Sequential statistical analysis after Wald assessed the information value and prognostic thresholds of the indices tabulated as the risk factors for autonomic dysfunction in children and adolescents. The results will be used in regular screening check-ups for the early detection of autonomic dysfunction in children, adolescents and for working out the preventive measures against a number of psychosomatic disorders in adults.

Adolescent↗
PubMed1985

[Clinical aspects of vegetative dysregulation during the pubertal period].

A clinico-populational study of the functional status of the vegetative nervous system in 5291 children of the puberal age made it possible, along with the so-called subclinical vegetative lability (a physiological phasic state of the neurovegetative apparatuses during puberty), to identify in 43.9% of cases clinicall manifestations of vegetative disregulation: the syndrome of clinically demonstrable vegetative lability and different syndromes of vegetative dysfunction. The frequency of vegetative disorders was correlated with sex and urban or rural living conditions of patients whereas their intensity largely depended on the level of functional activity of neuroendocrine apparatuses in the period of puberty.

Adolescent↗
PubMed1983

[Echoencephalographic indices in healthy children and age].

Five hundred and ninety-three healthy children and teenagers aged 2 to 20 years were studied echoencephalographically. Cranial anthropometry was also performed. Mean values of echoencephalographic findings for each age group were identified. These indices may be used in the future as a normal baseline helpful in the diagnosis of intracranial pathology. The age-related dynamic changes of the following parameters were examined: distances to the median complex (M-echo), echo-signal from the lateral ventricle opposite to the hemisphere being studied (ventricle echo--B-echo) to the final complex, as well as the width of ventricle III and the ventricular index. Age-associated correlation between echoencephalographic and anthropometric parameters was elucidated.

Adolescent↗
PubMed1983

[Electroencephalographic studies in children and adolescents with cerebral vegetative disorders].

The results of an echoencephalographic study of 403 children and adolescents aged 8 to 20 years, suffering from different vegetative disorders are presented. On the average, 49% of the patients examined showed a statistically significant dilatation of ventricle III of the brain. Echoencephalography is demonstrated to be a valuable method in the diagnosis of cerebral vegetative impairments in children and adolescents.

Adolescent↗
PubMed1981

[ABO blood group system and vegetative-vascular disorders in children].

The distribution of the blood groups of the ABO system was examined in 159 children with various vegetovascular disorders. The results were compared with the data on the distribution of the same blood groups in local population. A statistically significant (P less than 0.001) prevalence of persons with blood group A(II) and a lesser number of persons with blood group O(I) was revealed among the patients. It is assumed that the blood group A(II) is an unfavourable factor for children with vegetovascular disorders. An analogy is drawn with various diseases and cerebral circulation impairments in adults in whom a number of authors consider the blood group A(II) to be a risk factor.

ABO Blood-Group System↗
PubMed1980

[Clinico-electrophysiologic and pathomorphologic characteristics of congenital paralysis of the arms].

Results of clinical, electrophysiological and roentgenological examinations of 36 children with natal paralyses of the arms are analyzed. In two cases a postmortem verification was carried out. The data obtained suggest that natal damages to the humeral plexus structures and the spinal cord radicles underlie these paralyses. Only in individual cases characterized with a grave damage to the cervical division of the vertebral column spinal cord traumas may be responsible. The low-frequency activity of the affected muscles on an EMG is not yet a proof of a gross primary affection of the spinal cord, and cannot serve as a criterion for localizing the damage to the peripheral motor neuron. The bioelectrical activity changes should be interpreted with consideration of the complex of clinical symptoms.

Birth Injuries↗
PubMed1979

[Influence of the vertebral nerve on several indices of cerebral blood flow].

The dogs and rabbits, electrical stimulation of the vertebral nerve reduced the pressure in the carotid and the vertebral--basillar arterial systems leaving the general arterial pressure unaltered. Simultaneously the values of pH, PO2 PCO2, and buffer bases decreased in the blood flowing from the dog brain. The data obtained suggest that stimulation of the vertebral nerve reduces the blood flow in the vertebral artery which, in conditions of occlusion of the contralateral vertebral artery, leads to the circulatory regional hypoxia of the brain.

Animals↗
PubMed1977

[Means of experimentally reproducing the vertebral artery syndrome].

The authors suggest a method of reproducing the syndrome of vertebral artery in a chronic experiment on dogs. Two horse-shoe electrodes connected with the wires exteriorised to the posterior surface of the neck are introduced and fixed under general anesthesia at the selected level into the vertebral artery. Rheographic and thermometric shifts occurring in response to the electrode stimulation and to dosaged electrical stimulation were studied.

Animals↗