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Biomedical subjects

M E Seligman

Publications and source records attributed to M E Seligman.

At least 19 recordsLinked to original sources
PubMed1992

Predictors and consequences of childhood depressive symptoms: a 5-year longitudinal study.

A 5-year longitudinal study investigated the interrelationships among children's experiences of depressive symptoms, negative life events, explanatory style, and helplessness behaviors in social and achievement situations. The results revealed that early in childhood, negative events, but not explanatory style, predicted depressive symptoms; later in childhood, a pessimistic explanatory style emerged as a significant predictor of depressive symptoms, alone and in conjunction with negative events. When children suffered periods of depression, their explanatory styles not only deteriorated but remained pessimistic even after their depression subsided, presumably putting them at risk for future episodes of depression. Some children seem repeatedly prone to depressive symptoms over periods of at least 2 years. Depressed children consistently showed helpless behaviors in social and achievement settings.

Achievement↗
PubMed1991

Explanatory style and cell-mediated immunity in elderly men and women.

Correlated pessimistic explanatory style--the belief that negative events are caused by internal, stable, and global factors--with lowered immunocompetence in a sample of 26 older adults. Two measures of cell-mediated immunity--T-helper cell/T-suppressor cell ratio and T-lymphocyte response to mitogen challenge--were lower in individuals with a pessimistic style, controlling for the influence of current health, depression, medication, recent weight change, sleep, and alcohol use. A relative increase in the percentage of T-suppressor cells seemed to underlie this immunosuppression. Although the mechanism by which explanatory style might influence immune function remains unknown, we speculate that a pessimistic style might be an important psychological risk factor--at least among older people--in the early course of certain immune-mediated diseases.

Aged↗
PubMed1989

Assessing explanatory style: the content analysis of verbatim explanations and the Attributional Style Questionnaire.

We compare two methods of assessing explanatory style--the content analysis of verbatim explanations (CAVE) and the Attributional Style Questionnaire (ASQ). The CAVE technique is a method that allows the researcher to analyze any naturally occurring verbatim materials for explanatory style. This technique permits the measurement of various populations that are unwilling or unable to take the ASQ. We administered the ASQ and Beck Depression Inventory (BDI) to 169 undergraduates and content analyzed the written causes on the ASQ for explanatory style by the CAVE technique. The CAVE technique correlated 0.71 with the ASQ (P less than 0.0001, n = 159) and -0.36 with BDI (P less than 0.0001, n = 159). The ASQ correlated -0.51 with the BDI (P less than 0.0001, n = 160). Both the CAVE technique and the ASQ seem to be valid devices for assessing explanatory style.

Adult↗
PubMed1989

Explanatory style across the life span: evidence for stability over 52 years.

Analyzed explanatory style across the life span. 30 Ss whose average age was 72 responded to questions about their current life and provided diaries or letters written in their youth, an average of 52 years earlier. A blind content analysis of explanatory style derived from these 2 sources revealed that explanatory style for negative events was stable throughout adult life (r = .54, p less than .002). In contrast, there appeared to be no stability of explanatory style for positive events between the same 2 time periods. These results suggest that explanatory style for negative events may persist across the life span and may constitute an enduring risk factor for depression, low achievement, and physical illness.

Adaptation, Psychological↗
PubMed1989

Self-serving biases in causal attributions as a function of altered activation asymmetry.

The method of induced lateral orientation of attention was selectively used to alter the balance of activation of the brain toward one cerebral hemisphere or the other. The goal was to extend earlier research which showed greater personal optimism for future events during left hemisphere activation to the related phenomenon of causal attributions. As predicted, there was significantly greater self-serving bias (attribution of internal, stable, and global causes for positive events and external, unstable, and specific causes for negative events) during left hemisphere than during right hemisphere relative activation. This was true for achievement but not for affiliation events. These effects were explained in terms of the increased positive affect which correlates with greater left hemisphere activation. Selective activation of the left hemisphere as a means temporarily to alter attributional style was discussed as a possible tool in the treatment of learned helplessness and depression.

Adolescent↗
PubMed1976

Learned helplessness, depression, and the attribution of failure.

Depressed and nondepressed college students received experience with solvable, unsolvable, or no discrimination problems. When later tested on a series of patterned anagrams, depressed groups performed worse than nondepressed groups, and unsolvable groups performed worse than solvable and control groups. As predicted by the learned helplessness model of depression, nondepressed subjects given unsolvable problems showed anagram deficits parallel to those found in naturally occurring depression. When depressed subjects attributed their failure to the difficulty of the problems rather than to their own incompetence, performance improved strikingly. So, failure in itself is apparently not sufficient to produce helplessness deficits in man, but failure that leads to a decreased belief in personal competence is sufficient.

Achievement↗
PubMed1975

Frustation and learned helplessness.

This article reports the transfer of learned helplessness from one aversive motivator, shock to another, frustration. In experiment 1, animals were trained to approach food in a runway and concomitantly exposed to either escapable, inescapable, or no shock in a different situation. Extinction was conducted in the runway, and subsequently the animals were tested for hurdle-jump escape from the frustrating goal box. Inescapably shocked rats failed to learn to hurdle-jump, whereas escapably or nonshocked animals learned the frustration escape response. Experiment 2 replicated the basic finidngs of Experiment 1 and showed transfer of learned helplessness from shock to frustration when no running response had been first acquired in the runway.

Animals↗
PubMed1975

Conditioned drinking as avoidance learning.

Blocking the robust conditioned drinking response following conditioning eliminates the isotonic procaine conditioned response (CR) but not the hypertonic procaine CR. Rats allowed to drink in the box during conditioning form a larger CR than rats merely injected with hypertonic or isotonic procain who are prevented from drinking in the box: response blocking during extinction eliminated if no experience of drinking water in the box occurs either during baseline or during conditioning. Blocking the drinking response, like blocking shock avoidance, markedly reduces its high resistance to extinction. This suggests that rats overdrink to avoid anticipated illness, and fail to extinguish partially because they are never exposed to the fact that no illness will occur even if no drinking occurs.

Animals↗
PubMed1975

Learned helplessness in the rat.

Four experiments attempted to produce behavior in the rat parallel to the behavior characteristic of learned helplessness in the dog. When rats received escapable, inescapable, or no shock and were later tested in jump-up escape, both inescapable and no-shock controls failed to escape. When bar pressing, rather than jumping up, was used as the tested escape response, fixed ratio (FR) 3 was interfered with by inescapable shock, but not lesser ratios. With FR-3, the no-shock control escaped well. Interference with escape was shown to be a function of the inescapability of shock and not shock per se: Rats that were "put through" and learned a prior jump-up escape did not become passive, but their yoked, inescapable partners did. Rats, as well as dogs, fail to escape shock as a function of prior inescapability, exhibiting learned helplessness.

Animals↗
PubMed1975

Learned helplessness in the rat: time course, immunization, and reversibility.

Rats, like dogs, fail to escape following exposure to inescapable shock. This failure to escape does not dissipate in time; rats fail to escape 5 min, 1 hr., 4 hr., 24 hr., and 1 wk. after receiving inescapable shock. Rats that first learned to jump up to escape were not retarded later at bar pressing to escape following inescapable shock. Failure to escape can be broken up by forcibly exposing the rat to an escape contingency. Therefore, the effects of inescapable shock in the rat parallel learned helplessness effects in the dog.

Animals↗