A minor Q-independent pathway for the expression of the late genes in bacteriophage lambda.
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Biomedical subjects
Publications and source records attributed to M Couturier.
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Potential determinants of bone mass were investigated in a group of 70 young females (mean age 26.6 years), daughters of women studied in premenopause. Nutritional data, leisure physical activity level, lifestyle habits as well as familial similarities were assessed. The daughters' bone mineral density (BMD), measured by dual-energy absorptiometry, was significantly correlated with their body mass index (BMI) (r = 0.22), dietary vitamin D intake (r = 0.19) and their mothers' BMD (r = 0.44). Multiple regression analysis indicated that only the mothers' BMD remained an independent predictor of bone mass. Mother-daughter correlations were also observed for body weight (r = 0.24), height (r = 0.39), BMI (r = 0.29), dietary calcium intake (r = 0.20), and calcium (r = 0.20) or vitamin D (r = 0.25) intakes from dairy products. Hence, these observations support the evidence that mothers' BMD is the strongest predictor of bone mass of young women in their third decade.
Virulence-associated plasmids pLT2, pIP1350, pIP1360 and pIP1370 coded for the fi+ character. These plasmids did not code for the synthesis of classical adhesins, aerobactin or colicins. Nor did they mediate resistance to the metals Hg, Te, As, Cd or flavomycin. Plasmids pLT2, pIP1350 and pIP1370 contained DNA sequences homologous to the incFIIA and incFIB replicons. Plasmid pIP1360 contained a single sequence homologous to incFIIA replicon.
The authors studied the vitamin A status of 79 children, 71 of whom had liver disease. Plasma levels indicate low vitamin A concentration only when they are below 10 micrograms/dl. Study of liver concentration is currently the reference to appreciate vitamin A status. These concentrations are low in children with cholestasis. The lower the liver concentration, the higher the percentage of retinol in the liver. The percentage of the various retinyl esters is however independent of liver concentrations. The authors propose a model for the regulation of vitamin A metabolism within 4 pools.
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