Cell-surface changes of phosphate-deficient Escherichia coli as characterized by increased stability and decreased concanavalin A-agglutinability of the spheroplast.
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Biomedical subjects
Publications and source records attributed to M Arisawa.
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In Escherichia coli mutants deficient in DNA polymerase I, newly replicated short DNA is joined at about 10% of the rate in the wild-type strains. It is postulated that DNA polymerase I normally functions in filling gaps between the nascent short segments synthesized by the replication complex. Possible implications of the finding are discussed in relation to other abnormal properties of these mutants.
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