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Biomedical subjects

L Stoenescu

Publications and source records attributed to L Stoenescu.

14 recordsLinked to original sources
PubMedDate not supplied

Mechanisms and significance of QRS macrovoltage in the absence of cardiac hypertrophy.

The electric stimulation of the lateral ventricular walls carried out in experiments on dog heart (open-thorax), as well as the analysis of the clinical, radiological and electrocardiographic data recorded in 462 cases with QRS macrovoltage led to the following conclusions: a) in 22% of the cases (hospital cardiologic examinations) this anomaly cannot be accounted for either by age, blood pressure or cardiac hypertrophy; b) a temporarily perturbed development of the ventricular depolarization, i.e. a "jerky" depolarization, not stagnant enough to produce an intraventricular block, may generate great negativity and positivity myocardial masses responsible for the appearance of large dipoles, namely of the increased QRS voltage; c) the coincident ischemia and macrovoltage of the QRS major wave, as well as the subsequent evolution of incipient CHD in a series of patients point to the hypothesis according to which the regional myocardial ischemia may induce a QRS macrovoltage by means of the above mechanism.

Adult↗
PubMedDate not supplied

QT alterations and cardiodynamic parameters in old myocardial infarction.

Prolonged QT interval in patients with old myocardial infarction was noticed as a marker of poor prognosis. The sensitivity of this functional electrocardiographic scar was augmented in 82 patients with old myocardial infarction by recording the recumbent and orthostatic electrocardiogram, phonocardiogram and carotid piezogram. The incidence of abnormally prolonged QT intervals and increased QT ratios was three times higher in orthostatic posture than in patients lying down. Abnormal QT intervals in standing position correlate also with abnormal PEP/L VET ratio and severe electrocardiographic alterations and may become a useful tool for a more accurate prognosis in patients with old myocardial infarction.

Aged↗
PubMedDate not supplied

Sympathetic hyperfunction syndrome.

Vegetative troubles induced by sympathetic hyperfunction are presented and an attempt is made to systematize them. Functional tachycardia or premature beats in neurocirculatory asthenia, symptoms of sympathetic irritation in cervical radicular syndrome, hyperkinetic syndrome with positive exercise test, long Q--T syndrome, the classic troubles of sympathetic origin in pheochromocytoma and "the vegetative storm" in cerebral damages or excessively stressful situations may be included in the sympathetic hyperfunction syndrome. Of greater clinical interest is the cervical radicular syndrome with sympathetic irritation, which is presented in details, with reappraisal of some morphofunctional data that must be taken into account in clinical reasoning.

Angina Pectoris, Variant↗
PubMedDate not supplied

Cardiodynamic and electrocardiographic alterations induced by blood sampling in normal and hypertensive donors.

The 12 routine electrocardiographic leads, apical phonocardiogram, piezocarotidogram, Korotkov blood pressure, sitting and standing, were recorded before and after 500 cc blood sampling in 20 hypertensive donors and 20 normals. The cardiodynamic (systolic time intervals) disturbances in hypertensive donors were more frequent and more persistent than those observed in the normotensive group, despite the favourable effect of the blood sampling on the blood pressure values. The most significant cardiodynamic alteration was the increase in the pre-ejection period/left ventricular ejection time--indicating a more reduced ejection fraction in the hypertensive group and pointing out that the improvement of left ventricular work by decreasing the blood pressure after blood sampling is less important than the hypoxic effect of blood loss.

Blood Donors↗
PubMedDate not supplied

Reappraisal of some problems concerning the genesis of heart sounds.

This paper sums up for clinical purposes the recent data and some of the authors' own observations concerning the genesis and variability of heart sounds. The importance of some factors in the "cardio-hemo-vasculo-thoracic" complex where the heart sounds are produced and transmitted, is especially emphasized; such are: acceleration and decelaration of the blood, rapidly variable shape and compliance of the cardiovascular container, turbulence phenomena, atrial and ventricular contraction and relaxation, closure and opening of cardiac valves. The role of some physiological or pathological changes in myocardial contractility and compliance in over- or underdamping of intracardiac vibrations generating heart sounds, is also analysed. Some examples of alteration of the 4th, 1st, 2nd and 3rd sound in the 3rd degree A--V block, nodal rhythm, short P--R, LBBB, aortic atherosclerosis, myocardial infarction, are presented.

Adult↗