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Biomedical subjects

L Russegger

Publications and source records attributed to L Russegger.

47 records · Page 3Linked to original sources
PubMed1987

[Problems in medical expert assessment in cerebral aneurysm ruptures].

On the basis of two cases, the question is discussed as to whether or not rupture of a cerebral aneurysm during working hours can be classed as an industrial accident. The possibility that raised blood pressure during an extraordinary, very atypical effort could be the cause of rupture is discussed in detail. The authors stress their opinion that--from neurosurgical view point--the rupture of a so-called congenital berry aneurysm can never be classed as a working accident but is absolutely fortuitous. Every event of daily life thought to be the cause of aneurysm rupture is completely coincidental.

Accidents, Occupational↗
PubMed1986

Neurosarcoid reaction in association with a ruptured ACA-aneurysm.

A case of neurosarcoid reaction presenting itself as a mass lesion associated with a ruptured ACA aneurysm is reported. Diagnosis was established by histological investigation of the resected "brain tumour". The pre-operative diagnostic problems, similar cases in the literature as well as the possible pathogenesis are discussed.

Cerebral Cortex↗
PubMed1985

Treatment of cerebral vasospasm following severe head injury with the calcium influx blocker nimodipine.

8 patients suffering from severe head injury (Glasgow Coma Scale score of 3-5) were treated with the calcium influx blocker nimodipine (2-3 mg/h) for a traumatic cerebral vasospasm. In every case the spasm involved the frontal region of the circle of Willis with the first two segments of the anterior and middle cerebral artery. Control angiograms, recorded in 6 patients, revealed a dilated or normalized lumen of the vessels that the first angiography had revealed to be spastic. The intracranial and the mean arterial pressure were not altered by nimodipine during simultaneous neurosurgical intensive care therapy (dexamethasone, mannitol, relaxation, and controlled respiration for 2-3 days). Monitoring of the somato-sensory evoked potentials (SEP) showed a distinct improvement of the cortical response within 7 days. One patient died of sepsis 8 days after the accident and one remained in a vegetative state. The remaining patients reached Glasgow Coma Scale scores of 13.0 +/- 1.1. within 8.6 +/- 2.2 days, the initial scores having been 4.1 +/- 0.8. One year after their accident all the patients were once again fully able to work. The survival quality was therefore better than that reported in the literature for patients in similar conditions. We conclude from these preliminary results that calcium influx blockers can be used successfully in the therapy of traumatic vasospasm and of severe head injuries. A study involving a larger number of patients is currently in progress.

Adolescent↗
PubMed1984

[Clinical experiences with nimodipine (Bay e 9736)].

In 27 patients, who suffered from SAH from a ruptured cerebral aneurysm direct operation and treatment with Nimodipine (Bay e 9736) was performed. Nimodipine was given intravenously over ten days (30 micrograms/kg bodyweight/hour) and thereafter orally over four days in diminishing dosages. There were no noteworth side effects. In comparison with a group of nine similar patients who were not given Nimodipine the study shows that Nimodipine is not able to reduce angiographic spasm or brain oedema in CT-scan. In spite of that the general recovery with Nimodipine was better than in the control group. The worse the initial neurological symptoms are, the more effective Nimodipine seems to be. The study shows that treatment should begin between the first and sixth day after SAH, at least two days before operation and at the latest two days after the onset of secondary spasm.

Calcium Channel Blockers↗
PubMed1984

[Intracerebellar hematomas; diagnosis and therapy].

This article deals with the diagnosis, therapy and prognosis of intracerebellar haematomas. The characteristic symptoms and the value of computer tomography for an early diagnosis and the surgical therapy are discussed. Prognosis and complications are analysed in comparison with other works. Guiding rules for a rapid diagnosis and the choice of the respective therapy are given and an early surgical therapy is recommended.

Adult↗
PubMed1983

[Tumors of the 3d ventricle, clinical aspects and treatment].

Our last 30 patients, who were operated upon 3rd-ventricle-tumors, are discussed on symptomatology, diagnosis and therapy. Depending on the localization we classify oral, basal and caudal tumors. The peak of these tumors is found in the first ten years of life. They are distributed as followed: spongioblastomas, ependymomas, pinealoma and other rare tumors. Mainsymptom is the sudden diffuse headache depending on this skull's position. The three groups show different symptomatic features, the oral one sepecially that headache described above. The symptoms of the caudal group are due to signs of raised intracranial pressure and content the typical syndrome of the lamina-quadrigemina. Basal tumors lead to diencephalic disturbances. The CT scan should be done as the first diagnostic step, eventually connected with ventriculography or ventriculotomography. In any case therapy should be started by implantation of an atrio-ventriculare shunt. If the disease is progressing an invasive procedure has to be done. Irradiation therapy is bound to a clear histologic diagnosis or to a clear inoperability. Our 5-years survival was 40% in average.

Adolescent↗
PubMed1982

[The lucid interval in epidural hematoma].

On the basis of 191 patients with epidural haematomas in an observation period of 15 years, an analysis of the patient group with non-classical symptoms was carried out. The opinions and theories regarding the generation of long-lasting lucid intervals found in the literature are mentioned and discussed on the basis of 3 typical examples. Owing to improved examination methods and early recognition, the mortality rate of the patients with epidural haematomas could be reduced from 54 per cent in the years 1961 to 1965 to about 10 per cent in the years 1975 to 1980.

Adolescent↗