Biomedical subjects
L M Halpern
Publications and source records attributed to L M Halpern.
Analgesics and other drugs for relief of pain.
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The effect of diphenylhydantoin on the activity of selected enzymes in chronic isolated cerebral cortex of cat.
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Alterations in enzyme activity and hyperexcitability in the chronic undercut cerebral cortex of monkey.
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Augmentation of cerebellar Purkinje cell discharge rate after diphenylhydantoin.
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Effects of diphenylhydantoin and other antiepileptic drugs on epileptiform activity and Purkinje cell discharge rates.
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Diphenylhydantoin: evidence for a central action.
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Choline acetylase and acetylcholine esterase changes in chronic isolated cerebral cortex of cat.
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Alterations in the activity of selected enzymes in the chronic isolated cerebral cortex of cat.
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Drug therapy. II. Treatment of drug misuse.
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Stabilization of excitable membrane by chronic administration of diphenylhydantoin.
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The hyperexcitable neuron as a model for the laboratory analysis of anticonvulsant drugs.
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Micro-iontophoretic studies. I. Ejection characteristics of a series of five-barrelled micro-iontophoresis pipettes.
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Microelectrode studies of the afferent connections and efferent projections of neurons in the sensorimotor cortex of the cat.
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Antidepressants. Tetrabenazine-antagonizing activity in a series of 5H-dibenzo [a,d]cycloheptene-5-propylamines.
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Gallamine triethiodide facilitation of local cortical excitability compared with other neuromuscular blocking agents.
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The effect of diphenylhydantoin on post tetanic excitability in isolated rabbit vagus.
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Flaxedil (gallamine triethiodide): evidence for a central action.
A central action of gallamine triethiodide has been demonstrated in cats with permanently implanted electrodes that permit direct repetitive stimulation and recording of afterdischarges from cerebral cortex. Systemically administered gallamine produced a consistent and reproducible augmentation of duration of afterdischarge at doses just sufficient to produce skeletal muscle paralysis. Simultaneous examination of expiratory carbon dioxide, blood pressure, body temperature, blood glucose, and direct cortical response to brief single stimuli failed to reveal any consistent peripheral change to which the centrally observed effecte Could be attributed.