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Biomedical subjects

L Ferguson

Publications and source records attributed to L Ferguson.

49 records · Page 3Linked to original sources

Determination of the ligand-binding characteristics of several tight-binding mutants of the lactose repressor protein.

Several tight-binding mutants of the lactose repressor protein have been characterized with respect to their fluorescence properties and their inducer, operator and nonspecific DNA-binding constants. The tryptophan fluorescence emission spectra for the mutants and the wild-type repressor are quite similar. However, alterations in the Stern-Volmer constants for iodide quenching of the tryptophans in the mutant proteins compared to wild-type suggest differences in the local environment or solvent accessibility for these amino acids in the tight-binding repressors. The inducer-binding affinities and association rate constants of the mutant proteins and protein-operator DNA fragment complexes are also altered compared to wild-type. The extents of these changes vary among the different mutant repressors. The nonspecific DNA-binding affinities of the mutant proteins are 2--3-fold greater than the wild-type repressor, and the affinities of the tight-binding proteins for a 29 base-pair operator DNA fragment are also increased, though to a varying extent depending upon the mutant. The phenotypic behavior of these proteins in vivo can be partially explained by these results obtained in vitro; however, it is likely that there are additional factors responsible for the tight-binding behavior of the proteins that were not detectable in these experiments.

Alleles↗

Eosinophilic granuloma of the second cervical vertebra.

The occurrence of eosinophilic granuloma in the body of the second cervical vertebra of a young adult male is presented. The necessity of making a histologic diagnosis and prevention of vertebral instability led to operative intervention. Treatment consisted of posterior fixation of the spinous processes of the second and third cervical vertebrae with size 0 wire. Through a transoral approach the lesion was curetted, and bone chips were placed in the vertebral defect. The patient remains well, and disease-free, after 36 months.

Adult↗

Acute hemorrhagic cerebellar infarction following angiography.

Cerebellar infarction following cerebral angiography is an unusual complication. A case is reported. The patient's condition deteriorated with each diagnostic study. Improvement occurred after cerebellar decompression and evacuation of an intracerebellar hematoma.

Cerebellar Diseases↗

Filling patterns in contrast ventriculography.

The filling patterns of a negative contrast material (air), a positive contrast water soluble material (Conray), and a positive contrast water insoluble material (Myodil) were examined in 60 normal ventriculograms. Using a scoring system developed for this study, Conray was found effective for outlining the ipsilateral (injected) lateral ventricle, the third ventricle, the aqueduct of Sylvius, and the fourth ventricle. Air was the most effective for the noninjected lateral ventricle, while Myodil was best for A-P demonstration of the aqueduct. The clinical implications of these findings are discussed.

Cerebral Ventriculography↗

The significance of tibial artery disease in the management of popliteal aneurysms.

The clinical, radiographic and histologic features of sixty-one popliteal aneurysms in 36 patients are reviewed. Twenty-seven aneurysms were thrombosed and presented with an acutely ischaemic limb or the sudden onset of severe claudication. Thirty-four patent aneurysms presented with either ischaemic ulceration or claudication due to tibial artery disease or were asymptomatic with normal distal pulses. Thrombosis made reconstruction difficult and at times required a femoro-tibial graft. In others reconstruction was not possible leading to amputation either as primary treatment or following failed revascularization. In patent aneurysms one or more tibial arteries were frequently occluded. It is postulated that obliterative atheromatous disease of the tibial vessels and the slow flow through the aneurysmal vessels is responsible for the high incidence of thrombosis, poor graft patency and a high amputation rate.

Aged↗