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Biomedical subjects

L Crevits

Publications and source records attributed to L Crevits.

41 records · Page 3Linked to original sources

Paralytic pontine exotropia in subarachnoid hemorrhage. A clinicopathological correlation.

A syndrome of combined right lateral gaze palsy and right internuclear ophthalmoplegia, followed by a left exotropia in a case of subarachnoid hemorrhage is presented. At necropsy a single small pontine end-zone infarction was found, involving the medial part of the right paramedian pontine reticular formation, the tectospinal tract and the ventral part of the right medial longitudinal fasciculus. The fila radicularia of the right abducens nerve passed through the necrotic area. The mechanism of these characteristic ocular motor disturbances in relation to the post mortem findings and the topopathogenesis of the pontine infarction are discussed.

Female↗

Parallel visual and memory processes.

We studied parallel processes: visual processes with the prosaccade, the no-saccade and the antisaccade task on the one hand and memory processes with the random tap task on the other hand. The random tap task is believed to be a pure interference task for the central executive component of working memory. The number of saccadic errors was found not to be influenced by taxing the central executive, while the latency times were significantly increased both in the prosaccade and in the antisaccade task. The effect seen in the antisaccade task was expected since it is a non-automatic activity under central executive control. Because the prosaccade task is an automatic activity, an effect of central executive load was not expected. As an explanation for our findings, we postulate that the prosaccade task is brought under willed control of the central executive.

Adolescent↗

Central pontine myelinolysis and its arterial blood supply.

A case of central pontine myelinolysis is described. The clinical, angiographic and post mortem findings suggest that among the associated disorders the oligemic shock combined with a local decreased flow in the basilar artery was the most important cause of this patholigical entity. The angioarchitecture findings in the brain stem, compared to these in 26 normal brains, confirm this hypothesis.

Alcoholism↗