Human placental alkaline phosphatase and acute lung injury.
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Biomedical subjects
Publications and source records attributed to L Bossaert.
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A new method to describe intensive care department performance is presented. The method is a complication of available administrative and medical data, completed with a severity of illness measure (Acute Physiology And Chronic Health Evaluation, APACHE) and the registration of nursing care intensity. The development of this latter patient stratification system (Intensive Care Activity Score, INCAS) is described. The performance of the method is demonstrated by a study of 200 consecutive admissions.
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To determine the occurrence of inhibition of pacing by sensing of myopotentials, forty-five patients with predominant pacemaker driven heart rhythm were studied. In eleven patients with a bipolar lead system myopotential inhibition was never observed. In the remaining group (34 patients) with unipolar pacemakers myopotential inhibition was demonstrated in fifteen patients. Three of them were symptomatic, two severely. In forty new implants a silicone-rubber insulating sheath was placed against the muscular side of the pacemaker. In none of these patients myopotential inhibition could be demonstrated. The authors conclude that myopotential inhibition occurs frequently in unipolar ventricular inhibited pacemakers, and can be prevented by insulating the muscular side of the anodal surface.
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Five cases of idiopathic orthostatic hypotension are presented. Physiological tests were performed in four cases which demonstrated that the lesion in the sympathetic system was most probably situated in the intermediolateral columns in three cases. In one case the lesion was localized on the efferent pathway either at the preganglionic or the postganglionic level. These four patients had also evidence of parasympathetic dysfunction. In the fifth case the clinical diagnosis of Shy and Drager syndrome was confirmed by pathological examination of the nervous system which revealed intermediolateral cell loss and the pathological findings commonly observed in the striato-nigral and olivo-ponto-cerebellar degenerations.
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