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Biomedical subjects

Ken Kuriki

Publications and source records attributed to Ken Kuriki.

5 recordsLinked to original sources

Successfully treated idiopathic rectosigmoid perforation 7 years after renal transplantation.

Colon perforation (CP) is still a critical complication after renal transplantation (RT), and idiopathic perforation is extremely rare. Here we describe a successfully treated case of idiopathic rectosigmoid perforation that occurred 7 years after RT. In our research this is the tenth reported case of idiopathic CP after RT and the second case that has occurred in the rectosigmoid. The patient was a 51-year-old Japanese male RT recipient still receiving immunosuppressive medication. He was admitted to the hospital for sudden onset of abdominal pain during defecation. Emergency laparotomy was performed 5 h after the onset, and a longitudinal 1.5 cm perforation with a clear margin was observed in the rectosigmoid, 8 cm above the peritoneal reflection. Hartmann's operation was performed. Macroscopic and histological examination did not reveal any specific findings that may have caused perforation, so the case was diagnosed as idiopathic rectosigmoid perforation.

Humans↗

Double gallbladder.

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Cholangiopancreatography, Endoscopic Retrograde↗

Observation of fluorescence in an optically pumped erbium-containing perfluorinated medium at 1.5 microm.

We report what is to our knowledge the first observation of erbium fluorescence at the optical communication wavelength of 1.5 microm in a perfluorinated medium. Using a Nd:YAG (355-nm) source, we observed fluorescence of the 4I(13/2) - 4I(15/2) transition with a peak wavelength of 1.535 microm and with a full width at half-maximum of 28 nm. These results can lead to new active devices that use all-fluorinated polymers.

Journal Article↗

Severe strongyloidiasis complicated by meningitis and hydrocephalus in an HTLV-1 carrier with increased proviral load.

We report a 47-year-old Japanese man who was a human T-cell leukemia virus type 1 (HTLV-1) carrier with strongyloidiasis, and who was born in an area endemic for both Strongyloides stercoralis ( S. stercoralis) and HTLV-1. He presented with edema of both legs. Laboratory examination on admission revealed hypoalbuminemia, and S. stercoralis rhabditiform larvae were found by stool microscopy. Purulent meningitis, which was suspected to be due to disseminated strongyloidiasis, developed during the first and second treatment for S. stercoralis infection. After the meningitis was alleviated, hydrocephalus with gait disturbance developed, and these features were attenuated by a ventriculo-peritoneal shunt. Impaired immunity and increased HTLV-1 proviral load, with an increased titer of HTLV-1 antibody, were observed in this patient. These results suggest that HTLV-1 proviral load and/or antibody titer of HTLV-1 can be used for the identification of carriers who are at increased risk of developing severe strongyloidiasis among those patients who are infected with both S. stercoralis and HTLV-1.

Animals↗