Cerebral metabolic changes during treatment of subacute cerebral infarction by alpha and beta adrenergic blockade with phenoxybenzamine and propranolol.
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Adult↗
Biomedical subjects
K Shimazu
Publications and source records attributed to K Shimazu.
[A case of spinal segmental myoclonus and propriospinal myoclonus: a neuroelectrophysiologic analysis].
A 37-year-old male patient with spinal segmental myoclonus and propriospinal myoclonus was described. He was admitted to our hospital because of paroxysmal axial myoclonus, which first appeared one month before. He denied any significant accident such as trauma or fever. Apart from myoclonus, no abnormal findings were observed by physical and neurological examinations, routine laboratory investigations and MRI of the cervical and thoracic spinal cords. The myoclonus consisted of continuous rhythmic contractions of the bilateral thoracal and abdominal muscles. Its frequency was approximately 0.3Hz. The clinical findings were typical of spinal segmental myoclonus. In addition, the myoclonus started in the thoracal muscles and frequently spread up to the neck muscles and down to the leg muscles. The myoclonus disappeared in sleep. Polymyography revealed the following findings: (1) The jerks were found on the bilateral axial muscles including sternocleidomastoid, biceps, triceps, pectoralis major, abdominal muscles and quadriceps. (2) Homologous muscles were activated synchronously. (3) The duration of bursts was variable ranging 100 to 400msec. (4) The jerks in the pectoralis muscle preceded those in other muscles. The latencies of the jerks in the other muscles increased with their distance from pectoralis, based on the linear regression analysis of the onset of jerks in the various muscles. (5) The jerks were induced by tapping or electrical stimulation anywhere on the body including the face, but not by flash or sound. From the above polymyographical findings, the myoclonus seems to originate in the T3 spinal cord and slowly up and down the spinal cord at 0.6-1.6 m/sec, suggesting that it is mediated by the propriospinal tract.
Adult↗
A comparison of the results of A-C bypass grafting in collateral and non-collateral groups.
Ninety patients who had aorto-coronary bypass grafting were divided into two groups: a collateral group, which had coronary arterial stenosis or occlusion with collateral circulation, and a non-collateral group, which had coronary arterial stenosis or occlusion without collateral circulation. The number of coronary arteries visualized through collateral circulation in coronary angiograms (CAG) was 32, left anterior descending arteries (LAD) 17, right coronary arteries (RCA) 11, and left circumflex arteries (LCX) 4. The results of A-C bypass grafting in the collateral and non-collateral groups were compared. Surgical mortality was 0% in the collateral group, and 5.4% in the non-collateral group. The differences in graft patency and graft flow between the two groups were not statistically significant. However, left ventricular ejection fraction and myocardial perfusion, which was estimated by thallium-201 myocardial perfusion scintigram, were significantly improved after A-C bypass in the collateral group. Although the coronary arteries visualized through collateral vessels seemed too narrow to undergo graft anastomosis, they were, in fact, large enough. A-C bypass grafting was achieved with more satisfactory results in the collateral group than in the non-collateral group.
Angina Pectoris↗