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Biomedical subjects

K Eliasson

Publications and source records attributed to K Eliasson.

45 records · Page 3Linked to original sources
PubMed1980

Increased sympathetic activity and blood pressure in young asymptomatic men with 'organic' T-wave aberrations in the electrocardiogram.

1. Twelve asymptomatic young men with 'primary' T-wave aberrations in the electrocardiogram (group T) and 13 matched controls were subjected to a mental stress test, isometric exercise and a cold pressor test. 2. Plasma catecholamines and haemodynamics were studied. 3. Group T had signs of increased sympathetic activity at rest and enhanced sympatho-adrenal reactivity during stress. 4. Systolic blood pressure was consistently elevated in group T. 5. The T-wave aberrations may be explained by the increased sympathetic activity. 6. Several similarities exist between group T and borderline hypertensive subjects.

Adolescent↗
PubMed1979

Sympatho-adrenal and cardiovascular response to mental stress and orthostatic provocation in latent hypertension.

1. Seven latent hypertensive patients and seven matched controls were subjected to standardized mental stress and orthostatic provocation. 2. Mental stress increased blood pressure by approximately 25%, heart rate by 25 beats/min, plasma glycerol by 50% and plasma cyclic AMP by 25% in both groups. Plasma glucose and renin activity were unchanged. Plasma noradrenaline and adrenaline were essentially unchanged during stress. 3. There was an insignificant tendency towards higher noradrenaline levels in latent hypertensive subjects and two of these subjects displayed an exaggerated noradrenaline response to standing. 4. Our results indicate that the physiological responses to mental stress are caused by selective neuronal activation, rather than by generalized sympatho-adrenal activation. Latent hypertension does not seem to be associated with adrenergic hyperactivity or receptor supersensitivity, except possibly in individual cases.

Adult↗
PubMed1979

Vasospastic phenomena in patients treated with beta-adrenoceptor blocking agents.

Twenty-one patients developed Raynaud's phenomenon during treatment with beta-adrenoceptor blocking agents. The vasospastic symptoms were obviously related to the treatment, but their pathogenesis is still controversial. A decrease in cardiac output might explain the phenomenon. According to our very preliminary data an alpha-adrenergic dominance caused by a direct effect on the peripheral circulation seems more probable. Vasospastic symptoms may arise after treatment with both cardioselective and non-selective beta blocking agents. Cautious prescription of beta blocking drugs to patients with preexisting peripheral vascular disease is recommended.

Adrenergic beta-Antagonists↗
PubMedDate not supplied

Cervical metastases following radical neck dissection that preserved the spinal accessory nerve.

In contrast to the original neck dissection technique, the spinal accessory nerve is routinely sacrificed in the so-called classical neck dissection. The benefit of this routine has never been documented; on the contrary, facts have accumulated that indicate that the nerve should be preserved. The results in this article serve to emphasize this opinion. Of 80 patients who underwent radical neck dissection that preserved the spinal accessory nerve, the potential for cure was not jeopardized in a single case.

Accessory Nerve↗
PubMedDate not supplied

Occurrence, duration and prognosis of unexpected accessory nerve paresis in radical neck dissection.

Injuries to the spinal accessory nerve in connection with radical neck dissection occur frequently, despite the preservation of the nerve. Although the surgeon was unaware of any serious lesion of the accessory nerve, a trapezius paresis of varying degree was observed in about 60% of the patients during convalescence. The shoulder function improved in most patients, but major paresis with loss of essential rotary and supportive functions of the trapezius muscle persisted in 17% of the patients operated on. No further improvement was seen 18 months after the surgical trauma.

Accessory Nerve Injuries↗