Combining epikeratoplasty and photorefractive keratectomy.
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Biomedical subjects
Publications and source records attributed to K D Teichmann.
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A simple endocapsular technique for implantation of an 8 mm disc lens is described. Its main features are a scleral three-plane incision, a vertical-stab capsulotomy, cortical hydrodissection, a closed chamber technique during placement of the intraocular lens in the capsular bag, implantation under air and fluid, wherever feasible, and optional anterior capsulectomy with a 27-gauge needle bent at the tip. This technique requires no complicated equipment and allows atraumatic implantation of the disc lens with maximum control of anterior chamber depth.
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In part A a brief overview of systemic side effects of ophthalmic drugs such as antibiotics, betablockers, adrenergics, mydriatics and carboanhydrase inhibitors is given. Part B deals with ocular side effects of common drugs such as antibiotics, tuberculostatics, antirheumatics, corticosteroids and phenothiazines.
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Congenital glaucoma is one of the main causes of blindness in children and is more common in Saudi Arabia than in Western countries, perhaps owing to the prevalence of consanguinity with families. Trabeculotomy has given poorer in Saudi Arabia than elsewhere. This study compared the results of trabeculotomy (67% success rate) with trabeculectomy (54% success rate), and reports fewer complications following trabeculotomy.
Lid splitting procedures have been described a long time ago. Despite repeated descriptions by various authors, the method is not widely known. We use a bare-tarsus technique, omitting any grafting. In addition, we found radial incisions at the nasal and temporal edges of the anterior lamella advantageous, as the often superimposed blepharospasm is abolished by the weakening of the tarsal part of the orbicularis muscle. Also, there is then less tendency for the recessed anterior lamella to creep back down to the lid margin.
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A new approach to in-the-bag insertion is described. It combines elements of previously used techniques and includes a D-shaped capsulotomy, a posterior chamber intraocular lens with polypropylene loops and one control tip, a Sinskey, reverse Maltzman, or similar hook, and a lens loop injector (a simplified version of the Bailey lens shooter).
A case of severe visual loss following a single dose of vincristine is described. The primary pathology was a spinal cord astrocytoma, grade II, with no intracranial extension. Visual loss after a single small dose of vincristine has never been reported. While peripheral neuropathy and cranial nerve involvement is common, optic neuropathy is extremely rare. The literature of vincristine optic nerve toxicity is reviewed. Although differential diagnostic alternatives can be considered in all described cases it is very likely that vincristine does cause severe visual loss in a small number of patients.
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A family is described in which unilateral retinal detachment and oradialysis occurred in four of the six members (the father and three of his five children); there was no record of previous trauma. None of the subjects is myopic nor is any of them suffering from degenerative changes of the fundus. In all four cases the detachment was observed when they were aged between 18 and 26. An autosomal-dominant mode of inheritance is suspected but cannot be regarded as proved until the third generation has been examined.
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Two cases are presented, one with a pure cranial compression injury, the other mainly with chest trauma. Both patients noted immediate unilateral blindness. Vision did not improve in either within four months. Fluorescein angiography was performed and showed arteriolar as well as venous damage, with occlusion of arterioles and venules. As the impact in morbus Purtscher is usually very brief, reflux cannot explain the fundus changes. More likely a pressure wave is the cause of vessel damage with subsequent infiltration of blood, or plasma, into the wall of the vessel and obliteration of the lumen; it is also a cause of rupture of capillaries and hemorrhage. In traumatic asphyxia, on the contrary, a sustained force leads to reflux of blood and massive congestion with subsequent vessel damage and diapedesis. The prognosis in morbus Purtscher is often poor.
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Following a brief review of various interpretations of the disease and its treatment, the author's observations in 13 cases (18 eyes) treated during a period of ten years are reported. The data (Tables 2 and 3) are discussed with reference to the different forms of clinical manifestation of the disease (Table 4). Therapeutic aspects are outlined on the basis of the personal experience of the authors. If conservative measures (mydriasis, hyperosmotics, carboanhydrase inhibitors) fail to control ciliary block glaucoma, surgical procedures directed at the anterior hyaloid membrane are indicated. This structure is approached either via the pars plana or via the transpupillary route after lens extraction. In some cases lens extraction alone was sufficient.
The various causes of elevated intraocular pressure following glaucoma surgery are described. One has to differentiate between failures after peripheral iridectomy (Table 1) and those following filtering interventions. The clinical picture of a postoperative narrow-angle glaucoma must be analyzed carefully; it may be due to pupillary block, plateau-iris syndrome or even to a ciliary block (malignant glaucoma). The failure of filtering procedures may be due to mechanisms blocking the trephination opening, cicatrization of the conjunctiva or a ciliary block. The diagnosis criteria of the different conditions and the respective therapeutic measures are outlined.