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Biomedical subjects

J Roger

Publications and source records attributed to J Roger.

At least 109 records · Page 6Linked to original sources
PubMed1978

[Extension of infiltrating cancer of the stomach into the transverse colon (author's transl)].

The extension of infiltrating stomach cancer through the gastrocolic ligament into the transverse colon appears to be relatively common. The radiological appearance is characteristic, with more or less widespread, but shallow, extrinsic prints with irregular contours typical of malignant tumours. Its gastric origin is strongly suggested by its position of the superior border of the transverse colon. The authors report 6 observations in which the value of this very characteristic radiological abnormality of the colon to diagnose primary stomach cancer was confirmed. Radiological appearances are not very specific in primary stomach cancer and interpretation is often difficult; endoscopy or even biopsies are often inconclusive.

Adult↗
PubMed1977

[Treatment of corneal ulcers by hydrophilic lenses pre-soaked in proteolytic hydrolysates of the corneal stroma (author's transl)].

The faculty of confering to hydrophilic contact lenses the properties of corneal bandage made unable to swell has been investigated by saturating them with calf's corneal hydrolysate. The absorption and storage of the proteolytic hydrolysate (properties and collagenasic hydrolysate's technic of obtention is mentioned) by hydrophilic contact lenses are studied. The treatment of some corneal ulcers is related for 27 eyes from 20 patients. The results appear more suitable for torpic ulcers than for mechanical ulcers.

Contact Lenses, Hydrophilic↗
PubMed1977

[Partial epilepsy with complex symptoms and atrophic occipito-parietal lesions. Value of tachoencephalographic examination].

Eleven of the first 500 epileptics examined by axial tomography presented pseudo-porencephalic dilation (cavitary atrophy) of an occipital horn, associated or not with more extensive ventricular dilation. This lesion was found in 2.2% of all the epileptics and in 5% of the epileptics under the age of 20 in this group, a high incidence when compared with that of lesions of the same type located elsewhere. Clinical investigation and E.E.G. carried out on these 11 children showed that they were suffering from either hemiphegia or hemiparesis (8 out of 11 cases) and hemianopsia (7 out of 11 cases), and had serious epilepsy with partial fits (6 cases), usual temporal lobe epilepsy (5 cases), generalized secondary epilepsy (3 cases) or epilepsy defying classification (2 cases). These cases are compared with those recently published by Remillard, Ethier and Anderman. These authors consider that these cerebral lesions are the result of peri-natal thrombosis of the posterior cerebral artery. But in our group of cases, angiographic results do not appear to justify this claim.

Adolescent↗
PubMed1977

[Tomodensitometric study of cerebral accidents causing acute hemiplegia in children].

Computerized Axial Tomography (C.A.T.) easily distinguishes between the two types of cerebral accident responsible for the two major categories of acute hemiplegia in childhood. 1) In hemiplegias which develop in association with hemiclonic status epilepticus (H. H. and H. H. E. syndromes), in the majority of cases there is an appearance of cortico-sub-cortical atrophy involving the whole hemisphere contralateral to the hemiplegia. This atrophy develops following oedema of the hemisphere which accompanies the initial status epilepticus and which is clearly shown by a very early T.A.C. 2) In congenital or acquired hemiplegias not associated with status epilepticus there is, in most cases, an appearance of cerebral infarction or, very rarely, haemorrhage. It is thus possible, from a physiopathogenic standpoint, to draw a clear distinction between these two major forms of acute infantile hemiplegia. The first (H. H. and H. H. E. syndromes) usually result from hemispheric atrophy which develops in association with a unilateral or predominantly lateral episode of status epilepticus, whilst the second group are usually the result of cerebral infarction.

Adolescent↗
PubMed1975

[Atypical juvenile neurolipidosis. Ultrastructural study of a cerebral biopsy].

Electron-microscopic findings on a brain biopsy in a 9-year-old male affected by juvenile lipidosis are reported. Two types of neuronal cytosomes were discribed: lamellar concentric bodies, resembling membranous cytoplasmic bodies, and polymorphous complex bodies, unit membrane bound including various material, mostly resembling lipofuscin. The same storage was found in glial and endothelial cells. Neurochemical data failed to pin-point any specific abnormalities. The present case cannot be classified in the category of neuronal ceroid-lipofuscinosis but seems similar to certain reported cases of atypicalneurolipidosis.

Bone Marrow↗