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Biomedical subjects

J Limodin

Publications and source records attributed to J Limodin.

9 recordsLinked to original sources
PubMed2004

[The presentation of central pontine myelinolysis with a left upper monoplegia unassociated with hyponatremia or malnutrition].

Arising via an unidentified pathogenic mechanism, central pontine myelinolysis affects the central portion of the base of the pons. Rapid correction of hyponatremia is a frequent cause. The main symptoms are spastic tetraparesis, pseudobulbar paralysis and locked-in syndrome. We report a case of central pontine myelinolysis without hyponatremia, revealed by left upper monoplegia in a 40-Year-old female alcoholic patient without malnutrition symptom. Central pontine myelinolysis occurred consecutive to a suicide attempt with multiple drugs and alcohol. The diagnosis was confirmed by brain magnetic resonance imaging. This case illustrates the paucisymptomatic clinical characteristics of central ponine myelinolysis in patients without electrolyte and nutritional disorders.

Adult↗
PubMed1987

[Cerebral x-ray computed tomography and scintigraphy. Value of the combination of these investigative methods in ischemic pathology].

In 15 patients with a recent ischaemic cerebral vascular accident computerized tomography (CT) of the brain was normal whereas scintigraphy, performed at the same time, clearly showed the ischaemic area. These discordant findings may be interpreted as false-negative CT results. Factors such as the time elapsed between the cerebral accident and the CT examination, and the use or non-use of an intravenous contrast injection are discussed. It seems that the key factor is the proximity of the ischaemic lesion to the surface of the brain: cortical necrosis is undetectable at CT but distinctly visible at scintigraphy.

Adult↗
PubMed1987

Periodic alternating gaze: a case without any hemispheric lesion.

A clinicopathological study of a patient with periodic alternating gaze (PAG) is presented. PAG is usually associated with bilateral cerebral hemispheric ischemia. In our case, the anatomical lesion is very limited, restricted to the crus cerebri. The anatomical pathways involved and the mechanism of PAG are discussed.

Brain Stem↗
PubMed1987

[Slow, periodic alternating ping-pong gaze and Cheyne-Stokes respiration].

Periodic alternating ping pong gaze (PAPPG) represent a particular entity among abnormal ocular movements sometimes observed during coma. They usually result from a widespread bilateral hemispheric ischemia. A case of PAPPG is reported that presents a twofold particularity: the lesion responsible for the abnormal movement, although eventually bilateral, was initially unilateral, and the PAPPG were intermittent and associated with a Cheyne-Stokes respiration. This latter finding requires reconsideration of the nature of the stimulus responsible for this curious abnormal ocular movement.

Aged↗
PubMed1985

[Cerebral activation during pursuit eye movements: localization by the dichotic listening test].

The level of cerebral hemispheres activation is studied here during pursuit eye movement by means of a dichotic listening test. Visual pursuit, whatever the direction, clearly activates the right hemisphere; this is probably related to the phenomenon of attention. Each hemisphere is activated by contralateral pursuit eye movement, which suggests that each hemisphere plays a role in pursuit eye movement towards that direction.

Adult↗
PubMed1985

[Frontal syndrome of progressive supranuclear palsy].

Neuropsychological findings in 10 clinical cases of progressive supranuclear palsy are presented. Patients were aged 52 to 80 and the duration of their illness was 1 to 5 years. Severe psychological disturbances e.g. depression or outbursts of irritability were prominent features in 5 patients. Formal neuropsychological testing disclosed mental slowing, impaired attention, reduced verbal fluency and elaborated linguistic abilities, poor abstract thinking and reasoning, mild to moderate memory loss, dynamic apraxia, grasping, motor impersistence, imitation and utilization behaviour. The clinical similarities between these neuropsychological changes and frontal lobe syndromes, together with the lack of cortical involvement, suggest that the "dementia" of progressive supranuclear palsy could be explained by deactivation of the frontal cortex by subcortical lesions. The special importance of pallidal and mesencephalic reticular involvement in the syndrome is hypothetized.

Aged↗
PubMed1983

Ocular bobbing: abnormal eye movement or eye movement's abnormality?

Ocular bobbing is classified as an abnormal eye movement, resulting from pathological neuronal activity of the brain stem after bilateral pontine lesions. Clinical oculographic study of 5 patients suffering from ocular bobbing shows that upwards voluntary eye movements are abnormal. We suggest that ocular bobbing should not to be regarded as an abnormal eye movement, but, rather, as the only residual movement of patients which are totally deprived of both horizontal and upward movements.

Arterial Occlusive Diseases↗