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Biomedical subjects

J Kusakari

Publications and source records attributed to J Kusakari.

At least 109 records · Page 6Linked to original sources
PubMed1981

The effect of transient anoxia upon the cochlear potentials.

To contribute to the analysis of the electrocochleographical findings in humans, the reversibility of the cochlear potentials (the AP, CM, and SP) after transient anoxia was examined using 24 albino guinea pigs. The durations of anoxia ranged from 5 to 120 min, and the AP, CM and SP were examined one hour after restoration of the blood supply. The mildest form of cochlear damage after transient anoxia was the disappearance of the L-part of the AP and the severest was the complete abolition of these potentials. The severity of the damage closely correlated with the duration of anoxia. The AP was poorer in reversibility than the CM and the SP. These results were analysed and discussed in the light of literature on clinical and basic studies.

Animals↗
PubMed1981

ABR audiometry in the diagnosis of cerebellopontine angle tumors.

Auditory brain-stem responses (ABR) examination with simultaneous lobe-vertex (L-V) and membrane-vertex cm-v) recording was performed in 34 ears with cerebellopontine angle tumors. The detectability of wave I was better in M-V recording than in L-V recording, whereas L-V recording exhibited better detectability of wave V. ABR waveforms were classified into three groups, and the relations between the waveforms and the size of tumor or the degree of hearing loss were analyzed. Positive finding in ABR were obtained in 25 out of 34 ears in the present study. It was stressed that this dual recording in ABR was extremely useful in the diagnosis of cerebellopontine angle tumors.

Adolescent↗
PubMed1981

The inner ear dysfunction in hemodialysis patients.

Hearing and vestibular tests were performed in 229 patients who were treated with hemodialysis (H.D.) and the following results were obtained. (1) One hundred and six patients exhibited bilateral hearing loss and 31 showed unilaterally impaired hearing. (2) Vestibular dysfunction was found in 83 patients. (3) Those who were impaired both in the cochlea and the vestibular organ were 60. (4) No correlations were observed between the occurrence rate of the inner ear dysfunction and Hct, BUN and serum creatinine levels. (5) The results in the present study did not suggest the possibility that the hearing loss was progressively getting worse during the period of H.D. therapy. Based on these results, the possible etiological factors in the inner ear dysfunction were discussed.

Adult↗
PubMed1980

Study of a simultaneous lobe-vertex and membrane-vertex recording technique in auditory brainstem response.

The study of a simultaneous lobe-vertex and membrane-vertex recording in auditory brainstem response was performed in normal adults and in patients suffering from brainstem disorders. It was demonstrated that in normal subjects the detectability of waves I and V depends on the site of the recording electrodes and there is no statistical difference between the two recording methods in the latencies of the two waves. We reported 3 typical cases that illustrate the value of this recording method in the detection of the cochlear nerve or the brainstem disorders.

Acoustic Stimulation↗
PubMed1980

Reversibility of the endolymphatic potential after transient anoxia.

The effect of transient local anoxia (10 to 120 min) upon the endolymphatic potential (abbr. EP) was observed in 30 normal guinea pigs. The recovery of the EP was complete in the animal subjected to 10 min anoxia and almost complete in 20 min anoxia. When the duration of anoxia was 30 min or longer, the recovery of the potential was incomplete, and the longer the duration of anoxia, the lower the level of the EP after reoxygenation. These results were discussed in relation to the possible role of a transient anoxia upon sudden deafness.

Action Potentials↗
PubMed1979

Effect of ethacrynic acid upon the peripheral vestibular nystagmus.

The effect of ethacrynic acid (ETA) upon pendular rotation nystagmus and paralytic nystagmus was examined using 61 guinea pigs. 100 mg/kg ETA reduced these nystagmus but 30 mg/kg ETA had on effect. Galvanic nystagmus, Bechterew's compensatory nystagmus, OKN and EEG were not affected by 100 ng/kg ETA. These results are highly suggestive that the inhibition of the peripheral vestibular nystagmus by ETA is mainly due to the reduction of the ampullar endolymphatic potential.

Action Potentials↗