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Biomedical subjects

J K Torng

Publications and source records attributed to J K Torng.

16 recordsLinked to original sources
PubMed1992

The contralateral variation of peak systolic velocity in common carotid arteries.

In order to evaluate the contralateral variation of the peak systolic velocity (Vmax) in common carotid arteries (CCAs), 9 clinically healthy persons (Group A), 11 patients with simple goiter (Group B) and 16 patients with diffuse toxic goiter (Graves' disease, Group C) were referred for radionuclide scanning, duplex Doppler ultrasonography (duplex US) and determination of serum level of the thyroid hormones (T3, T4, T3U) by radioimmunoassay. In Group A, Vmax of right CCA was 98.8 +/- 34.5 cm/sec and that of left CCA was 113.4 +/- 28.9 cm/sec; the left CCA had a higher blood flow velocity than the right one in normal subjects (p less than 0.01). However, the left and right CCA Vmaxes did not significantly differ to each other in Group B (93.5 +/- 21.4 cm/sec vs 98.6 +/- 21.4 cm/sec, p greater than 0.05) and Group C (146.9 +/- 29.3 cm/sec vs 149.9 +/- 28.9 cm/sec, p greater than 0.05). We conclude that there is a contralateral difference of blood flow velocity of common carotid arteries with the left value higher than the right one in healthy persons and this should be kept in mind in evaluation of thyroid functional and carotid arterial diseases.

Adult↗
PubMed1991

The peak systolic velocity of the common carotid artery and superior thyroid artery as an indicator of thyroid function.

We investigated the efficacy of color duplex ultrasound in the detection of the thyroid's functional state on 132 consecutive patients referred for morphological examination of the thyroid gland. The patients were classified into 3 groups: Group A (euthyroid, n = 108), Group B (diffuse toxic goiter, n = 16) and Group C (hyperthyroidism on medical treatment for 1 to 4 months, n = 8), according to the serum thyroid hormones' determination and the patient's clinical history. The peak systolic velocity (Vmax) of the bilateral common carotid arteries (CCAs) and superior thyroid arteries (STAs) were measured. The Vmax of CCAs was 90.33 +/- 23.13 cm/sec (mean +/- standard deviation), 148.44 +/- 28.97 cm/sec and 84.75 +/- 13.58 cm/sec in Groups A, B and C. The hyperthyroid patients had a significantly higher Vmax of CCAs than the euthyroid subjects (p less than 0.001) and those hyperthyroid patients receiving medical treatment (p less than 0.001). The Vmax of STAs was 32.4 +/- 13.49 cm/sec for the non-nodular lobes and 38.78 +/- 13.76 cm/sec for the nodular lobes in the euthyroid subjects. There was no difference between the non-nodular and nodular lobes. The Vmax of STAs was 91.69 +/- 32.02 cm/sec and 62.75 +/- 18.19 cm/sec for Groups B and C; both were higher than the euthyroid (p less than 0.001 and p less than 0.05 respectively). Patients with hyperthyroidism had a lower Vmax of STAs after medical treatment (Group B vs Group C, p less than 0.01). It is our conclusion that cases of hyperthyroidism have a higher volume of blood flow of both the common carotid arteries and thyroid arteries than the euthyroid cases.(ABSTRACT TRUNCATED AT 250 WORDS)

Adult↗
PubMedDate not supplied

Anterior pituitary functions in patients with uremia tested by stimulation with four combined hypothalamic releasing hormones.

Seven cases with uremia (6 men, 1 woman, mean age = 55.6 +/- 2.2 years) were studied with four combined hypothalamic releasing hormones (corticotropin-releasing hormone, CRH; luteinizing hormone-releasing hormone, LHRH; thyrotropin-releasing hormone, TRH; and growth hormone-releasing hormone, GHRH) for assessment of anterior pituitary functions. The mean basal levels of corticotropin (ACTH, 22.4 +/- 5.2 pg/ml), thyrotropin (TSH, 2.4 +/- 0.6 microU/ml), and follicle stimulating hormone (FSH, 26.0 +/- 3.4 mIU/ml) in uremic patients were not significantly different from those (34.0 +/- 3.5 pg/ml, 2.0 +/- 0.4 microU/ml, and 23.2 +/- 6.4 mIU/ml) of controls (5 men, 1 woman, mean age = 54 +/- 2.5 years), but the ACTH and TSH responses to the releasing hormones were significantly lower than those of the controls. The mean basal levels of luteinizing hormone (LH, 70.7 +/- 16.3 mIU/ml), cortisol (9.8 +/- 1.2 micrograms/dl) and prolactin (109.3 +/- 23.2 ng/ml) in uremic patients were significantly higher than those of normals (27.3 +/- 6.6 mIU/ml, 6.5 +/- 0.7 micrograms/dl and 15.7 +/- 3.4 ng/ml), while suppressed LH, cortisol and prolactin responses to the releasing hormones were observed in the uremic group. The mean basal growth hormone (GH) level in uremic patients (3.1 +/- 0.4 ng/ml) was not significantly different from that (2.8 +/- 0.7 ng/ml) of normals, but the GH response to the releasing hormones was significantly higher than that of controls. These results show pituitary dysfunction, such as blunted ACTH, TSH, LH and prolactin response, exists in uremic patients.(ABSTRACT TRUNCATED AT 250 WORDS)

Adrenocorticotropic Hormone↗