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Biomedical subjects

J Floquet

Publications and source records attributed to J Floquet.

At least 73 records · Page 4Linked to original sources
PubMed1984

[Chemodectoma of nerve X. Value and interest of the concept of the diffuse paraganglionic system].

Tenth nerve chemodectoma is one of the rarest varieties of cervicocephalic paragangliomas, far behind tumors developed from the carotid body. Based on findings in three patients, one reported previously, clinical and biological features of these formations are discussed. Recent data and those from personal studies are exposed, and arguments presented enabling the concept to be defended of a diffuse paraganglionic system in relation with the vagus nerve.

Adult↗
PubMed1982

[Familial presenile dementia: Gerstmann-Sträussler-Scheinker's syndrome (author's transl)].

A similar affection has developed in eight members from four generations of a family living in the Alsace. The disease is characterized by the onset of a pyramidal, pseudobulbar syndrome and dementia during the third or fourth decade of life. The outcome is fatal after a mean period of three years. Cerebral biopsies in three cases have demonstrated multicentric amyloid plaques differing from senile plaques. Clinical and pathological findings are similar to those currently reported in the literature as being typical of Gerstmann-Sträussler-Scheinker's syndrome. The affection appears as a separate entity: the multicentric plaques, clinical symptomatology, pyramidal or pseudobulbar, cerebellar syndromes, usually preceding dementia, age of onset, course, and familial character or the disorder distinguish it among presenile dementias. Its clinical profile and course are very similar to that of familial cases of Alzheimer's disease, some of which are probably cases of Gerstmann-Strässler-Scheinker's syndrome. Transmission to animals, though inconstant, places it within the group of transmissible dementias among kuru, Creutzfeldt-Jakob's, and familial forms of Alzheimer's disease. The familial nature of the affection and the variability of clinical and pathological features in the same family illustrate the complex relationships between hosts and pathogenic agents in the clinicopathological expression of a disease.

Adult↗
PubMed1981

[Hodgkin's disease with muscular involvement. 2 cases (author's transl)].

The authors report on two cases of Hodgkin's disease with muscular involvement. This exceptional complication occurred during the terminal phase of a stage IV disease in one case and during remission of a stage III disease in the other case, where it responded to combined chemotherapy. Muscular lesions in Hodgkin's disease may be due to extension from adjacent tissues or from dissemination through the blood stream. They do not necessarily indicate a more severe prognosis.

Adolescent↗
PubMed1980

[A case of non-bony chondroma of the finger (author's transl)].

A rather unusual case of a cartilaginous tumor of the soft tissues, without bone connections, is reported. The tumor was a chondroma developing from the extensor tendon sheath. The general characteristics of non-bony chondromas are described and similar observations reported in the published literature reviewed. Problems raised by differential diagnosis by radiological examination are discussed.

Aged↗
PubMed1979

[Right ventricular dysplasia; a study in 7 cases].

Seven new cases of right ventricular dysplasia are described, five of which were complicated by ventricular tachycardia. The frontiers of this syndrome are unclear and it is uncertain whether it should be considered as a separate disease entity, or as a minor form of "paper thin" right ventricle or Uhl's disease.

Adult↗
PubMed1979

[Is the primary lesion in Behcet's disease a venous "tropism" vascularity? (author's transl)].

The authors studied eight cases of Behcet's disease, all of which were characterized by different degrees of phlebitis disorders. They also reviewed the anatomical and clinical features of a similar type, reported in the published literature. They stress the frequency and severity of changes in the veins in the localization of the various symptomatic lesions of the affection. From these results, they conclude that the primary lesion in this disease could be a phenomenon of vascularity of mainly venous-"tropism" which would explain the proteiform clinical features of Behcet's aphthosis.

Behcet Syndrome↗