[Unilateral posterior-column syndrome due to intramedullary neurinoma. Imitative homolateral synkinesias].
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Biomedical subjects
Publications and source records attributed to J Cambier.
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Abnormal ocular movements were observed in a patient with myoclonic encephalopathy secondary to branchial carcinoma. Clinical, cinematographic, and oculographic studies demonstrated the presence of rapid, large amplitude, conjugated, horizontal movements which occurred in bursts. They had a tapered appearance and were triggered by fixing vision and jerking movements, while they were abolished by rest and closing the eyelids. These features are similar to those seen in macrosaccadic oscillations. Other types of oscillating ocular movements are discussed as well as the physiopathological features of this disorder.
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A case of familial cerebellar ataxia is reported during the course of which horizontal ocular movements were slow, and "viscous". The electro-oculographic study showed complete abolition of horizontal jerks, induced by either voluntary, automatic (optokinetic nystagmus), or reflex (vestibular) stimuli. Slow movements, however, were normal, and voluntary vertical jerks were conserved while being slightly slowed towards the top. The physiopathological interpretation of these facts remains a point for discussion. Results from animal experiments and neuropathological studies suggest that the abolition of jerks in such cases is probably due to loss of facilitating influences from the cerebral cortex and central grey nuclei.