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Biomedical subjects

J Cadilhac

Publications and source records attributed to J Cadilhac.

At least 91 records · Page 5Linked to original sources
PubMedDate not supplied

[The E.E.G. and E.M.G. findings in sudanophilic leucodystrophy (author's transl)].

Four patients with infantile familial sudanophilic leucodystrophy confirmed histologically and biochemically were investigated by E.E.G. and E.M.G. The E.E.G. showed a severe, non-specific slow wave abnormality, of moderate voltage and disorganized in 2 cases, with some critical' tonic' discharge. E.M.G. study was normal in relaxed muscle; peripheral nerve stimulation, however, revealed marked slowing of motor conduction velocities favouring a segmental demyelinisation process, which was ultimately confirmed histologically. This study underlines the value of investigation of infantile encephalopathies.

Cerebral Cortex↗
PubMedDate not supplied

[Myotonic seizures; the action of curare or related compounds on myotonia (author's transl)].

Myotonic seizures with apnea are alarming but temporary incidents occurring during curaization of a myotonic patient. Experience with regional curaization in myotonia shows that it is induced only by suxamethonium and it is only an exaggeration of the fasciculations produced by this compound. In contrast, however, competitive curare compounds have no effect on myotonia. The myotonic seizure, which is specific to myotonia, is radically different from the other respiratory accidents common to muscular dystrophies.

Adolescent↗
PubMedDate not supplied

[HLA and amyotrophic lateral sclerosis (author's transl)].

52 patients with amyotrophic lateral sclerosis and 300 normal subjects were tested for 33 HLA-A, B, C antigens. We did not observe any statistically significant difference in antigenic frequencies between the two groups. The absence of association with HLA in amyotrophic lateral sclerosis is a distinction with multiple sclerosis, which is associated with several HLA antigens.

Amyotrophic Lateral Sclerosis↗