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Biomedical subjects

J C Honet

Publications and source records attributed to J C Honet.

36 records · Page 2Linked to original sources
PubMed1977

Carpal tunnel syndrome or trigger finger associated with neck injury in automobile accidents.

Among 450 patients examined for neck pain following automobile accidents, carpal tunnel syndrome (CTS) was diagnosed in four and trigger finger (TF) in three. All seven patients sustained cervical sprains and were drivers or passengers in automobiles which received impacts from the rear. All seven patients reported the onset of hand symptoms during or shortly after the accident. This unusual occurrence, not previously reported, appears to be related to the events which occurred at the time of the accident. The mechanism of injury for CTS is hypothesized to be median nerve compression by momentary acute hyperflexion or hyperextension of the wrist(s), while tightly grasping the steering wheel or bracing for impact. The cause of TF is suspected to be direct trauma to the flexor tendon and sheath by compression against bony prominences when the finger is hyperflexed or hyperextended. This can occur during the accident while forcefully grasping the steering wheel with acutely hyperflexed fingers.

Accidents, Traffic↗
PubMed1976

Cervical radiculitis: treatment and results in 82 patients.

Depending on the severity of the clinical problem, three categories of treatment--home, outpatient and hospital--were utilized in 82 patients whose conditions met the strict criteria for cervical radiculitis. Details of the clinical picture, laboratory findings, electromyographic examinations, category and results of treatment are detailed. The follow-up results in 59 patients contacted one to two years after the initial treatment are also included. Eighty percent of the patients had good to excellent results when treated with conservative therapy during the initial episode whereas 71% continued to have good to excellent results after one to two years. Seventeen percent of those patients who were followed required a laminectomy.

Adult↗
PubMed1976

Neuropathy in the upper extremity after open-heart surgery.

Eleven patients with upper-extremity neurological abnormalities underwent open-heart surgery performed through a median sternotomy incision. Seven of the 11 patients were referred in the routine manner to evaluate and treat the neurological problem. The remaining four were part of a consecutively studied group of 11 patients examined prospectively to determine the possible presence of abnormalities. Two of these four patients were asymptomatic. All lesions could be postulated to occur within the brachial plexus, the most common area being the median cord, but lesions were also noted in the posterior and lateral cords and upper trunk. The etiology of the problem appears to be stretching injury of the brachial plexus from retraction of the sternum, which in turn causes retroclavicular displacement of the clavicle. However, it is possible that an ischemic neuropathy could result from intraarterial procedures in some of our patients. The possibility that neurologic deficit may occur in the upper extremity should be considered by physicians who may have the opportunity to evaluate patients who undergo open-heart surgery.

Adult↗
PubMed1975

Neurological abnormalities in the leg(s) after use of intraaortic balloon pump: report of six cases.

Six patients from a group of 39 who survived after treatment with the intraaortic balloon pump (lABP) had significant neurological deficits in one or both legs associated with the use of the lABP. The device was used in a group of 89 patients initially for cardiogenic shock but its use has been expanded for patients having the following conditions: preshock; severe congestive heart failure; refractory angina; and for those undergoing open-heart surgery. The six patients who had neurological sequelae had eight lABP insertions into the thoracic aorta through the femoral artery and had neurological abnormalities and/or electromyographic abnormalities in nine lower extremities ranging from a foot drop to almost total paralysis of the lower extremity. The pathophysiology of the neurological deficit is postulated to be an obstruction to blood flow, or thromboemboli, in the femoral artery.

Adult↗