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Biomedical subjects

J Beurey

Publications and source records attributed to J Beurey.

At least 37 records · Page 2Linked to original sources
PubMed1984

[Livedo reticularis and cerebrovascular accidents].

The authors report the case of three middle-aged women admitted to the hospital because of cerebro-vascular accidents, in whom examination disclosed idiopathic livedo reticularis. This association has only been described recently, but does not seem fortuitous; some of the clinical manifestations are characteristic, but the histology is usually nonspecific and the basic abnormality remains unknown. The main problem is prognostic and therapeutic. In an attempt to prevent the neurological defects, tobacco and contraception should not be permitted to female patients showing widespread acquired livedo reticularis; high blood-pressure should be treated systematically.

Adult↗
PubMed1982

[Cervical Pyoderma gangrenosum: treatment by plasma exchanges].

The authors report one case of Pyoderma gangrenosum whose unusual cervical localization was particularly bothering. Besides, the patient, an 18-year-old girl, had clinical symptoms of Crohn's disease confirmed by radiological investigations and bowel pathology. A benign monoclonal gammapathy and an auto-immune haemolytic anaemia were also found. After four plasma exchanges Pyoderma healed rapidly, Crohn's disease stabilized and biological abnormalities disappeared. Comments and review of the literature are reported. It appears that such a case is quite exceptional, as far as clinical, biological data and therapy (plasma exchange) are concerned.

Adolescent↗
PubMed1982

[Heredity and prevention of atopy].

Although clinical studies have been supplying strong arguments in favour of a hereditary transmission of atopy for a long time, the latest works have not made it possible yet to specify the biologic support of this heredity. An obvious correlation, especially, has not been demonstrated between, on the one hand, atopic ground and HLA group and, on the other hand, between a high production of total IgE and HLA group. At present, the production of specific IgE only seems to be really subordinated to the HLA system; it is likely to be the expression of complex immunological disorders of a genetic determinism. Various environmental factors will contribute, on this predisposed ground, to the disclosure or to the worsening of the atopy. Some prevention of atopy may be undertaken. Extended breast feeding, eviction of some environmental nuisance and family affective balance thus represent useful preventive measures. The influence of the date of conception and of the pregnancy and delivery conditions does not appear to be insignificant; but, there, the prevention is more difficult to set up. Finally, the counting of the IgE in the cord blood could lead to the detection of high-risk newborns.

Breast Feeding↗
PubMed1982

[Treatment of tinea microsporum. Metabolic interference between phenobarbital and griseofulvin].

Two epileptic children treated with phenobarbital and suffering from tinea capitis, not responding to a long lasting treatment with Griseofulvin, healed only after phenobarbital has been discontinued. We ascribe our therapeutic problems to the metabolic interference of Griseofulvin with phenobarbital. We have measured the blood concentration of Griseofulvin several times in one of our 2 children and in a control of the same age living in the same dietary conditions and in the same conditions of Griseofulvin intake; these measurements confirm these data. The point of interest of this report is to draw attention on: -the pharmacologic hypotheses put forward to explain this drug interference, -the other factors of a bad therapeutic response to a treatment with Griseofulvin.

Child↗
PubMed1980

[HLA-DRw7 in psoriasis vulgaris].

40 unrelated patients with Psoriasis vulgaris were studied for their HLA-A, B and DRw antigen phenotypes. All underwent a skin biopsy to confirm diagnosis. Like most of the authors, we observed an increase in B13 and B17 antigens (17,50 and 25%) whereas in the healthy population the percentage was (5 and 6,02%) respectively. The strong increase in DRw7 suggests that psoriasis vulgaris was associated with DRw7 antigens, with an unbalanced linkage between the B13 and DRw7 antigens and between the B17 and DRw7 antigens.

Adult↗