Biomedical subjects

J Barmeyer

Publications and source records attributed to J Barmeyer.

At least 73 records · Page 4Linked to original sources

PubMed1977

[Heart size and left ventricular function in coronary artery disease. II. Heart volume, coronary morphology and laevocardiogram (author's transl)].

We have investigated the possible relationship between the radiogically determined cardiac volume and the coronary angiogram and laevocardiogram. There was no relationship between cardiac size and coronary angiogram. Independently from the number of coronary vessels involved, we found normal sized hearts in patients without ECG-evidence of myocardial infarction, and enlarged hearts in patients with ECG-evidence of myocardial infarction. There was a significant, though loose relationship between the cardiac volume and the endsystolic and enddiastolic volumes (r=0.73 and 0.55 respectively) and the ejection fraction (r=0.69) as determined by laevocardiography. The critical value of the cardiac size, about which one encounters an increased number of abnormal volume parameters, was the upper boundary of 1-SD. Using this value we found a specifity of 81% and a sensitivity of 74% for the heart volume as a predictor of a pathological ejection fraction. On the other hand, using the upper boundary of 2-SD as a critical value, there was a sensitivity of only 58%, but a specificity of 92%. Only 11% of the patients with a cardiac size in the lower range of normal or below had an ejection fraction below 50%. Therefore the radiologically determined heart size is a simple, in daily practive acceptable method to assess and follow up left ventricular function in coronary patients.

Adult↗

PubMed1976

[Substitution of the aortic value in florid bacterial endocarditis and severe aortic valvular insufficiency (author's transl)].

Urgent operation for replacement of the aortic valve for the correction of a severe, increasing hemodynamic impairment of the output of the heart and for the removal of the septic focus in florid bacterial aortic endocarditis must be carefully thought over. 3 of our own 7 patients died. The indication is to be made largely on hemodynamic grounds when the clinical symptoms are quite clear without preoperative catheter diagnosis. Preliminary specific treatment with antibiotics and intensive follow-up treatment is necessary. Preoperative identification of bacteria in the blood, just like identification of bacteria on the excised valve, is only possible in some of the cases.

Acute Disease↗

PubMed1976

[Clinical and hemodynamic aspects of myocardial diseases (author's transl)].

Definition, arrangement into primary and secondary forms and classification of myocardial diseases are used irregularly in the literature. The great variety of clinical and hemodynamic parameters are shown with reference to our own patients and an attempt is made to arrange them in individual types. Followup examinations show that signs of congestive failure, heart size and other clinical and hemodynamic parameters may be considerably altered. For this reason it is proposed to describe myocardial diseases accoridng to clinical and hemodynamic parameters in order to obtain a closer insight into the pathomechanism of these diseases.

Cardiomegaly↗

PubMed1976

[Clinical course of arteriosclerosis of coronary and lower limb arteries (author's transl)].

The clinical course of arteriosclerosis was evaluated in 9 patients with coronary sclerosis and in 25 with peripheral arterial occlusive disease by means of repeat angiographies following primary angiography after 22.5 and 36.6 months, respectively. During the period of observation arteriosclerosis had progressed in all cases except in two patients with peripheral arterial occlusive disease. Despite intensive therapeutic efforts including control of risk factors, dietary care, guided or unguided exercise and the attempt to change unsatisfactory living conditions, progression of arteriosclerosis could not be interrupted. If treatment delayed progression of arteriosclerosis cannot be decided. All vascular areas were affected indiscriminately by progression and no particular localisation was preferred. Intensive exercise does not seem to delay progression of arteriosclerosis.

Angiography↗

PubMed1976