[Myofascial pain dysfunction syndrome. II].
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Biomedical subjects
Publications and source records attributed to I Marini.
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We studied the possibility of inducing a bronchospasm by a cold stimulus (0 degrees C) on the chest wall of normal subjects and those with bronchial lability. The stimulus is applied on areas in metameric connection with the bronchial tree. Modifications of MEF25, MEF50 and FEV1.0 before and after stimulation are surveyed. Positive bronchospasm indexes are pointed out in a large number of subjects with bronchial lability, while no obstructive bronchial response is found in normal subjects. The cold stimulus on the chest wall can therefore be considered as an aspecific stimulus producing bronchospasm. Some physiopathogenetic interpretations are suggested.
Muscular pain sensitivity after aerobic exercise was investigated in ten healthy men aged 20 to 30 years in four tests at five-day intervals to determine if previous aerobic work leads to a hyperalgesic status. The intensity of pain was recorded, by visual analog scale, every 30 seconds after the injection of 1 ml of 10% and of 20% sodium chloride hypertonic solution, both during basal conditions, and 1 ml of 10% sodium chloride hypertonic solution 1 and 60 minutes after 30 minutes of submaximal rectangular exercise. The injection of 10% sodium chloride solution 60 seconds after exercise gave rise to a clear increase in pain, similar to that induced by the 20% sodium chloride solution given during basal condition. This observation shows that submaximal exercise produces a hyperalgesic state in the active muscle. This hyperalgesia probably explains the clinical manifestation of latent algogenic triggers during physical activity.
Pain caused by muscular isometric contraction was investigated through use of the visual analog scale. The results demonstrate that the latency time and the time to reach maximum intensity are inversely proportional to the amount of the load; therefore increase of pain is linear. The high statistical significance of the results suggests that pain by isometric contraction could be a comparison test for somatic pain evaluation in spontaneous pathological conditions.
The subject of present research has been to study the effects of ischemia determining pain and cardiorespiratory modifications observed during and after isometric contraction of a quadriceps muscle subjected to a constant load. Therefore, in the same lower limb used for the previous test; an ischemia has been caused and protracted for the same amount of time. During and after the test the pain's intensity, VE, HR and VO2 have been noted. The obtained results have been subjected to statistic survey which has not proved significant variations of the studied cardiorespiratory parameters. Moreover pain has not arise during the test. A supposition that in those conditions only a little quantity of active metabolites can, be made. They conclude, therefore, that ischemia is a modest component in the cardiorespiratory modifications' determinism obtained with a test of isometric contraction of a quadriceps muscle subjected to a constant load.
On the aim to evaluate the influence of ischemia in the modifications of some cardiorespiratory parameters during and after an isometric contraction of he quadriceps muscle subjected to a constant load, the authors have been studied the behaviour of VE, VO2, HR and intensity of pain on subject which has had ischemia on both lower limbs. The results have been used for a statistic survey. During the test any VO2 meaningful variations have not been observed, while a VE and HR linear increase has been noticed. Furthermore pain has not appeared. Absence of pain, HR decrease and fast VE and VO2 increase, has been observed after the test. They conclude that a large area of ischemia for a sufficiently long time produce a small variations in some of the cardiorespiratory parameters studied. It is so prove that muscular work and not the ischemia is the principal component in the production of metabolites that cause the pain and also the cardiorespiratory modifications.
Patients affected with juvenile rheumatoid arthritis present clear radiological alterations of the condyles of the temporomandibular joint (TMJ) due to the inflammatory process, with a prevalence ranging from 17 percent to 63 percent. This work is a comparison between a group of subjects with juvenile rheumatoid arthritis presenting signs and symptoms of TMT dysfunction and a group of the same age not suffering from any rheumatic morbidity, but presenting an open bite > 3 mm. The aim is to verify whether the open bite can induce an alteration in mandibular function comparable to that observed in juvenile rheumatoid arthritis. Statistically significant difference is found in the juvenile rheumatoid arthritis group only regarding spontaneous and provoked muscle pain and the lateral opening deviations of the mouth, but mostly the two groups seem alike and present the same stomatognathic pattern.
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