[Protective effect of hypophysectomy in so-called "dystrophic tissue calcification"].
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Calcification, Physiologic↗
Biomedical subjects
H SELYE
Publications and source records attributed to H SELYE.
[Alteration of indium poisoning by parathyroid gland hormone and dihydrotachysterol].
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Dihydrotachysterol↗
[On a calcifying form of anaphylactoid inflammation].
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Albumins↗
Calcinosis induced by lead acetate.
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Calcification, Physiologic↗
Experimental production of cholangitis and pancreatitis by ferric oxide saccharate (Fe-OS).
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Cholangitis↗
A calciphylactic syndrome affecting the skeletal muscles, the salivary glands and the eyes.
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Calcification, Physiologic↗
Prevention by ferric dextran of the topical calcification induced by direct calcifiers.
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Calcification, Physiologic↗
Inhibition by hypophysectomy of organ lesions normally produced by parathyroid hormone or dihydrotachysterol.
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Calcification, Physiologic↗
Selective uterine or pancreatic lesions by calciphylactic system.
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Anaphylaxis↗
Effects of vitamin E on cardiomuscular calciphylactic lesions.
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Calcification, Physiologic↗
Acute polyarthritis produced by lipopolysaccharides in rats.
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Animals↗
Cutaneous molt induced by calciphylaxis in the rat.
A molt, conducive to the loss and subsequent replacement of all cutaneous layers, can be induced by topical "calciphylaxis" in the rat. This is accomplished by sensitization with dihydrotachysterol followed by challenge with egg white or ferric dextran.
Animals↗
Calcification of the parathyroids induced by calciphylaxis.
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Calcification, Physiologic↗
Production of calciphylactic facial, oesophageal, and mediastinal lesions by combined treatment with dihydrotachysterol and thorium dioxide.
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Calcification, Physiologic↗
An experimental model of "dermatomyositis" induced by calciphylaxis.
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Calcification, Physiologic↗
Excessive stimulation of salivary gland growth by isoproterenol.
In the rat, chronic treatment with isoproterenol can cause a selective growth of the salivary glands to approximately five times their normal size within 17 days. This enlargement is principally due to mitotic proliferation and hypertrophy of the parenchymatous cells.
Animals↗
Stress and renal function in relation to the hyalinizing and calcifying lesions of connective tissue.
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Amyloidosis↗
Systemic and topical factors involved in the production of experimental cutaneous calcinosis.
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Calcification, Physiologic↗