The results of this study, plus those of a previous study, show that a layer of CaOH lining the floor of the tooth cavity will protect the dental pulp against toxic irritants inherent to an UV light-polymerized cavity liner and a UV light-polymerized composite restorative resin. This study also showed that a layer of CaOH lining the floor of the cavity will protect the dental pulp against acid irritants inherent in the enamel etching solution. It is recommended to restorative dentistry that all primary dentin within clinically accessible areas of the cavity preparation be covered with a layer of CaOH before the enamel is etched with the acid-conditioning solution. Precautionary protection of primary dentin in this manner affords a margin of safety should the conditioning solution inadvertently spill onto the primary dentin during the enamel-etching procedure. Second, if the conditioning solution does accidentally spill onto the previously placed protective layer of CaOH during the enamel-etching procedure, then it logically follows that this acid-contaminated layer of CaOH should be mechanically removed and a new layer of CaOH that completely lines at least the floor of the tooth cavity should be placed before the resins are applied.

Acid Etching, Dental↗

PubMedDate not supplied

Vital root resection: a conservative procedure for abutment teeth.

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Dental Abutments↗

PubMedDate not supplied

A histological comparison of direct pulp capping agents in primary canines.

The purpose of this study was to evaluate the pulpal response to four calcium hydroxide preparations in primary teeth. Data indicated the calcium hydroxide-saline paste induced more inflammation and produced a greater zone of mummification than the three commercial preparations studied. Unintentional deep impaction of the medicament or dentinal chips increased inflammation for all categories. All medicaments produced a superficial necrosis (mummification) of the pulp. The greatest degree of mummification was seen with the calcium hydroxide-saline paste. The calcium hydroxide-saline paste produced a thick dentinal bridge, located deeper in the pulp when compared with the commercial agents. Commercial agents produced narrower but sufficiently high-quality dentinal bridges. This study indicates that direct pulp capping of exposures in primary teeth is a viable procedure. The newer commercial agents may be preferred, as they produce minimal inflammation, satisfactory dentinal bridging, and preservation of most of the vital pulp tissue.

Calcium Hydroxide↗

PubMedDate not supplied

A comparison of two calcium hydroxide agents in direct pulp capping of primary teeth.

Twenty-two pairs of bilateral primary canines, indicated for extraction due to excessive crowding, were chosen to study pulpal response to direct pulp capping with two calcium hydroxide medicaments (Life and Dycal). Following mechanical exposure of the pulp and direct pulp capping with calcium hydroxide, the teeth were extracted after either seven or sixty-three days to determine both the immediate and long-term pulpal response to the medicaments. The results indicate no statistical difference between the two materials in terms of biological response. Favorable results as to degree of pulpal inflammation and matrix formation indicate direct pulp capping using calcium hydroxide in primary teeth with no pulpal inflammation may be a viable procedure.

Calcium Hydroxide↗

PubMedDate not supplied

The effect of age and everyday exercise on steady-state plasma digoxin concentrations.

Age-related physiologic changes may significantly alter the disposition and pharmacologic characteristics of many drugs. The elderly are the most frequent users of digoxin because of increased prevalence of atrial fibrillation and congestive heart failure. This study was conducted to confirm the decrease in digoxin concentrations during exercise, to determine if age is a factor in this decrease, and to explore the difference between chronologic age and physiologic age. Eighteen men age 50-85 years were treated with digoxin for more than 1 month before enrolling and had serum digoxin concentrations of 0.4-2.0 micrograms/L. They were evaluated during a 3-hour period in the morning (A.M. dose withheld). Blood samples were obtained every 10 minutes during sequential 60-minute periods of rest (phase I), walking (phase II), and rest (phase III). There were no significant differences in mean concentration between phases II and I (p < 0.76), phases III and phase I (p < 0.70), or phases II and III (p < 0.37). The effect of age was positively correlated with the mean concentration of phase II but was not statistically significant (p < 0.62). Statistically significant correlations were seen only between the exercise phase and serum albumin and Mini-Mental Status Examination scores. We conclude that exercise has minimal, if any, clinically relevant effects on plasma digoxin concentrations. Increasing chronologic age has no influence on a decrease in the concentrations with exercise; a younger physiologic age may play a role.

Age Factors↗