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Biomedical subjects

H Cotta

Publications and source records attributed to H Cotta.

At least 37 records · Page 2Linked to original sources
PubMed1982

[The post-traumatic frozen shoulder].

The post-traumatic frozen shoulder is a sickness which develops relatively frequently following even small injuries of the shoulder joint. The particular anatomic conditions of this joint are the cause of its frequent lesions. After an injury typical pathological changes in function are observed. Most common injuries are distorsions, contusions, luxations and especially in increasing age the subcapital fractures of the humerus. Other injuries as there are rupture of the acromio-clavicular joint, fractures of the clavicle or scapula may also cause a frozen shoulder. The clinical examination is most important as it is the basis of the therapy. The current procedure consists of intensive physical therapy. If this treatment is not successful other ways must be discussed, e.g. mobilisation under anaesthesia. Operative treatment has a very strict indication. Medico-legal points of view of the frozen shoulder are difficult but a connection between injury and degenerative changes ought not to be denied in clear cases.

Adult↗
PubMed1981

[Comments on classification and terminology of congenital malformations in lower arm and hand (author's transl)].

It is the aim of this paper to draw attention to the problems facing the clinician when he tries to find a suitable nomenclature and classification of congenital malformations of the forearm and hand. The classification presented here takes into consideration the so-called Paris Nomenclature of 1969, as well as the systematisation of limb defects of the Working Group of the International Society for Prosthetics and Orthetics (commented upon by Henkel and al 1979), as well as the systematic organisation of malformations of the forearm and hand by Müller (1937), Werthemann (1952), Hopf (1959) and Witt, Cotta, Jäger (1966). On no account may this be regarded as a rigid and final setup. Efforts to achieve greater precision are still under way as before, with special emphasis on significant information supplied by teratology, embryology, genetics and biochemistry.

Arm↗
PubMed1979

[Physiology and pathology of the epiphyseal cartilage (author's transl)].

Knowledge of the physiology of the epiphyseal cartilage, respectively epiphyseal plate, is essential for an understanding of defective growth and abnormal modeling of the long bones. The epiphyseal cartilage develops from the embryonal, cartilaginous long bone structure. The histology of the epiphyseal cartilage is characterised by definable zones representing the individual differentiation steps from the reformation of cartilage to chondrolysis. Modeling of the ends of the long bones is also influenced by a transversal and longitudinal direction of growth in the epiphyseal cartilage. The intercellular substance mainly contains collagin, proteoglycanes and non-collagenic proteins. These macromolecules are compounded by means of physicochemical bonds and are responsible for the special mechanical qualities of the hyaline cartilage. The process of mineralisation at the base of the epiphyseal cartilage is an essential differentiating step for the ossification processes which take place in the metaphysis. Two pathogenetic principles at the epiphyseal cartilage appear to be important for the defective growth of the long bones. On the one hand, the flowing equilibrium between the differentiation steps of cartilage reformation, transformation of the hyaline cartilage into a mineralised cartilaginous tissue and chondrolysis is changed, whereas on the other hand the turnover of these differentiation steps is retarded or accelerated.

Animals↗
PubMed1977

[Acute traumatic cartilage damage. Experimental studies and their practical consequences].

Emphasis is placed on the etiology of posttraumatic cartilage damage based on recent experimental findings. The latter suggest that postinjury cartilage impairment cannot only be regarded as a consequence of incongruity of the joint surfaces after intraarticular fractures but that immobilization, primary lesions of the joint surface caused by synovial enzymes, and hemorrhagic effusion are also pathogenetically significant. Furthermore posttraumatic synovitis with its faulty composition of the synovial fluid plays an equally important role in this context. Finally, in summarizing therapeutic recommendations are presented.

Acute Disease↗
PubMed1976

[Late complications after cross-plate arthrodesis of the hip (author's transl)].

Cross-plate arthrodesis produces a problem of incongruence which cannot be solved by surgical skill Mechanical factors, not foreseeable before operation, produce an inevitable divergence of pulling and torsion forces with a high risk of losening of the implant and pseudarthrosis, particularly when the acetabulum is degenerated. The mechanical forces are shown in Fig. 6. The review of our cases shows that healing after cross-plate arthrodesis may follow three pathways.

Arthrodesis↗