Early behavioural effects of intraventricular administration of 6-hydroxydopamine in rat.
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Animals↗
Biomedical subjects
H C Fibiger
Publications and source records attributed to H C Fibiger.
Brain enzymes in amantadine-treated rats.
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Acetylcholinesterase↗
Enhanced stimulant properties of (+)-amphetamine after chronic reserpine treatment in the rat: mediation by hypophagia and weight loss.
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Analysis of Variance↗
The effect of para-chlorophenylalanine on aversion thresholds and reactivity to foot shock.
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Animals↗
Axonal transport in nigro-striatal and nigro-thalamic neurons: effects of medial forebrain bundle lesions and 6-hydroxydopamine.
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Animals↗
Neostriatal choline acetylase and cholinesterase following selective brain lesions.
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Acetylcholinesterase↗
Potentiation of amphetamine-induced arousal by starvation.
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Amphetamine↗
Differential development of caudate enzymes in the neonatal rat.
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Acetylcholinesterase↗
A biphasic action of central cholinergic stimulation on behavioral arousal in the rat.
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Animals↗
Effect of acute and chronic methamphetamine treatment on tyrosine hydroxylase activity in brain and adrenal medulla.
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Adrenal Medulla↗
The effect of para-chlorophenylalanine on spontaneous locomotor activity in the rat.
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5-Hydroxytryptophan↗
Effect of adrenal demedullation on starvation-induced behavioral arousal.
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Adrenal Glands↗
Aging and brain enzymes.
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Acetylcholinesterase↗
The effect of amantadine on spontaneous locomotor activity in the rat.
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Amantadine↗
Effects of insulin on spontaneous activity during food deprivation.
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Animals↗
Cholinergic modulation of adrenergic arousal in the developing rat.
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Age Factors↗
Ontogeny of adrenergic arousal and cholinergic inhibitory mechanisms in the rat.
With spontaneous activity as a measure of arousal, dose response curves were established for scopolamine and amphetamine administered to 10-, 15-, 20-, 25-, and 100-day-old rats. Amphetamine always increased activity, but scopolamine had no efect on younger rats, which suggests that adrenergic excitatory areas in the brainstem mature more rapidly than cholinergic inhibitory areas in the forebrain.
Aging↗
Deficits in instrumental responding after 6-hydroxydopamine lesions of the nigro-neostriatal dopaminergic projection.
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Animals↗