Peculiar myofilament changes near the intercalated disc in isoproterenol-induced cardiac muscle cell injury.
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Animals↗
Biomedical subjects
G Rona
Publications and source records attributed to G Rona.
In memoriam. Eors Bajusz M.D., Ph.D. 1926-1973.
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Canada↗
Studies on protein passage through arterial endothelium. 3. Effect of blood pressure levels on the passage of fine structural protein tracers through rat arterial endothelium.
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Animals↗
[Microcirculatory aspect of myocardial lesions induced by catecholamine infusion. Ultrastructural study with diffusion tracers. I. Isoproterenol].
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Animals↗
Fibrin as a natural tracer in cardiac muscle cell injury.
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Animals↗
Pathogenesis of isoproterenol-induced myocardial alterations: functional and morphological correlates.
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Animals↗
Pathogenesis and pathology of cobalt cardiomyopathy.
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Animals↗
Ultrastructural changes in isoproterenol-induced atrial necrosis.
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Animals↗
[Myocardial lesions induced by isoproterenol with the aid of diffusion tracers].
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Animals↗
Ultrastructural studies on myocardial fibrin deposition in experimental hypertension.
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Animals↗
Studies on the mechanism of increased myocardial resistance.
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Animals↗
Early alterations of the cardiac muscle cells in isoproterenol-induced necrosis.
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Animals↗
Myocardial resistance to isoprenaline in rats: variations with time.
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Animals↗
The protective effect of estrogens against spontaneous pancratic islet and renal changes in aging male rats.
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Aging↗
Electron microscopic cytochemical study of glutamic oxalacetic transaminase activity in ischemic myocardium.
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Animals↗
Fine structure of extra-nodal transitional cardiocytes in rat left atrium.
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Animals↗
Experimental aspects of cobalt cardiomyopathy.
All chambers of the heart are affected in experimental cobalt cardiomyopathy, with atrial predilection. The primary morphological alteration is mitochondrial damage that possibly reflects an enzymatic block of oxidative decarboxylation at pyruvate and ketogluterate levels. In acute cobalt toxicity chelation of calcium may also be a contributory factor, resulting in deficient utilization of high-energy phosphates. Experimental cobalt cardiomyopathy requires preconditioning factors: protein deficiency appears to be one of them. Vegetative polypoid endocarditis is an important accompaniment in the model used, suggesting that in rats on a protein deficient diet cobalt produced endothelial damage in addition to a cardiocyte injury.
Animals↗
Functional and fine structural heterogeneity of atrial cardiocytes.
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Cell Nucleus↗