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Biomedical subjects

G Reichel

Publications and source records attributed to G Reichel.

At least 55 records · Page 3Linked to original sources
PubMed1981

[Visceral diabetic polyneuropathy - quantitative determination of respiratory cardiac arrhythmia].

In 100 insulin-dependent diabetics, the degree of the respiratory cardiac arrhythmia as a measure of the participation of the N. vagus in the polyneuropathy is determined by means of the triggered cross-correlation function. The impairment of the respiratory cardiac arrhythmia shows statistical relations to the electroneurographic changes of somatic nerves, to the duration of diabetes and to the occurrence of microangiopathies.

Adolescent↗
PubMed1981

[The prognostic significance of electromyographic and electroneurographic results in Landry-Guillain-Barré-Strohl polyradiculoneuritis].

On the basis of an analysis of electromyographic and electroneurographic findings in patients with polyradiculoneuritis Landry - Guillain - Barré - Strohl courses of the diseases with different prognoses are found. In pure demyelinisation processes, a rapid and complete restitution can be expected. The regression of the pareses, however, will show a delayed, frequently an incomplete onset when electrophysiologically and axon loss can be proved.

Adolescent↗
PubMed1980

[Electromyography and electroneurography in peripheral nerve lesions. Part I].

First, the possible forms of the lesion of peripheral nerves are defined in the form of a survey: pressure damage (neurapraxia), axon interruption with maintained envelope structures (axonotmesis) and complete separation of the nerve (neurotmesis). From these, the various prognostic characteristics of a nerve lesion can be derived. An neurological examination is not sufficient for a thorough study of these phenomena. A particularly valuable aid is offered by the neuroelectrodiagnostic methods of electromyography and electroneurography. The technical fundamentals of these are briefly dealt with by the authors and indications are derived from them for the use of the two methods. In case of the open nerve lesion, the methods are used for a demonstration of intact nerve fibres, and, in the later phase, for the proof of returning spontaneous activity as well as the degree of the progress of a reinnervation. As far as chronic pressure damage is concerned, for example in bottle-necks, information regarding the seat and the degree of the nerve damage can be derived. And finally, EMG and ENG permit to make statements about the segmental height of radicular damage; this is necessary in case of plexus damage, but also for the demarcation of distal lesions. In the second part, the combination with the clinical practice and therapy will be dealt with in greater detail.

Electromyography↗
PubMed1979

[Diabetic polyneuropathy. 4. Synopsis of electroneurographic findings in diabetics].

Sensory conduction velocity of the median nerve, motor conduction velocity of both median and tibial nerves, and corresponding distal laterncies are sufficient parameters to establish the diagnosis of polyneuropathy almost with certainty. Considering these six parameters yielded in detection of peripheral nerve dysfunction in 22% of diabetic patients who were free from clinical signs of polyneuropathy. Electroneurographical findings in 340 out of 677 patients with diabetes mellitus were interpreted as evidence of segmental demyelination. Within this group there was the majority of patients with clinical signs of polyneuropathy and with subclinical signs of peripheral nerve dysfunction. There existed a positive correlation between signs of nerve dysfunction with angiopathy, age and duration of the disease. A second group consisting of 243 diabetics with signs of incipient segmental demyelination with or without signs of axonaal degeneration mainly included juvenile patients with a short duration of the disease and with a low frequency of angiopathy.

Adolescent↗
PubMed1979

[Method for studying spinal evoked potentials].

The authors describe a new method of examing evoked spinal potentials. Derivation is by means of unipolar needle electrodes which are ventrolaterally introduced into the cervical disk space as far as the posterior longitudinal ligament. Detection of a spinal potential following the stimulation of brachial and crural nerves allows to exclude the possibility of complete transverse lesion of the spinal cord or severing of the peripheral nerve or plexus.

Brachial Plexus↗
PubMed1979

[The diabetic polyneuropathy. I. Relation between impaired function in peripheral nerves and clinical findings].

789 patients with diabetes mellitus were studied by clinical and electroneurographical investigation. Motor and sensory conduction velocities of the median nerve and motor conduction velocity of the tibial nerve were determined. 86.1% of the patients suffered from juvenile diabetes, and 13.9% from maturity onset diabetes. Average duration of the disease was 9.5 years, average age of the patients was 26.7 years. Clinical signs of polyneuropathy were found in 19.1%. In 40.9% of the patients at least one of 3 conduction velocities was found to be delayed. Patients with clinical signs of polyneuropathy exhibited delayed nerve conduction velocities and delayed distal latencies. Diagnosis of polyneuropathy almost with certainty is possible by determining the three nerve conduction velocities and the three corresponding distal latencies. 22% of patients without clinical signs of polyneuropathy exhibited electroneurographical signs of impaired peripheral nerve function. Heredity, body weight, lipid metabolism, actual metabolic balance, and treatment were found to be without any significant influence on nerve conduction velocity.

Adult↗
PubMed1979

[The diabetic polyneuropathy. II. Polyneuropathy, angiopathy and nerve conduction velocity].

789 patients with diabetes mellitus were studied by clinical and electroneurographical examination. Motor conduction velocity of the median and the tibial nerve and sensory conduction of the median nerve were determined. 81.1% of the patients we suffering from diabetes which began in childhood or adolescence, 13.9% were suffering from maturity onset diabetes. Average duration of the disease was 9.5 years, average age was 26.7 years. Clinical signs of polyneuropathy were found in 19.1%. Typical findings were pain and paraesthesia, lack or abolition of triceps surae reflexes, impaired pallaesthesia on lower extremities. 48.3% of 151 patients with clinical signs of polyneuropathy were suffering from combined angiopathy, 32.5% from microangiopathy, 7.9% from macroangiopathy. Severity of complicating retinopathy and macroangio,athy were found to be correlated with polyneuropathy. 58.2% of 323 diabetics with at least one delayed nerve conduction velocity exhibited signs of angiopathy. In nearly 30% of children and adolescents after comparatively short duration of the disease at least one conduction velocity was delayed. In diabetic children and adolescents metabolic disturbances are assumed to cause peripheral nerve dysfunction.

Adolescent↗
PubMed1979

[Electromyographic examinations of the musculus sphincter urethrae in children with meningomyelocele].

Thirty children with neurogenic voiding attendant upon myelomeningoceles were examined, and for twenty-three children the electromyographic findings were compared with urodynamic measuring results. The authors conclude that an increase in subvesical resistance is due not to an increase of tonicity of somatic sphincter muscles but to a predominance of sympathetic over parasympathetic innervation.

Electromyography↗
PubMed1979

[Diabetic polyneuropathy. 3. Electroneurographic findings in diabetics and their relationships to age and duration of diabetes].

Nerve conduction velocities were determined in patients with diabetes mellitus: motor conduction of the median nerve in 778 patients, sensory conduction of the median nerve in 680 patients and motor conduction of the tibial nerve in 745 patients. In 40.9% out of 778 patients at least one of the three nerve conduction velocities were found within pathological ranges. 30.4% of 227 patients below 19 years of age in whom the duration of the disease did not exceed four years exhibited at least one delayed nerve conduction velocity. Clinical signs of polyneuropathy in children and in adolescents below 19 years of age are rare (0.6%). In contrast delayed nerve conduction velocities were found in 29.4%. Metabolic disturbance of peripheral nerve function is assumed to be responsible in these patients, for angiopathy in children and adolescents is very rare too.

Adolescent↗
PubMed1977

[The effect of roentgen rays on the transmembrane potential and on the electrophoretic mobility of polymorphonuclear granulocytes and FL-cells (author's transl)].

Two hours after X-irradiation at 37 degrees C in vitro at doses ranging from 50 rad to 20 krad the transmembrane potential and the surface charge of polymorphonuclear granulocytes and FL-cells showed a dose-dependent decrease. However, between two and four days after irradiation at 10 krad FL-cells revealed an increase in both transmembrane potential and surface charge. Small doses of ionizing radiation (e.g. 10 rad) resulted in aperiodically damped oszillations of membrane polarisation in leucocytes.

Cell Line↗