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Biomedical subjects

G Rau

Publications and source records attributed to G Rau.

At least 181 records · Page 10Linked to original sources
PubMed1980

Multi-electrode recording of Hoffman reflexes.

A multi-electrode system has been used to simultaneously record the H-reflex and M response from various locations on the gastrocnemius and soleus. With certain positions of the stimulating electrode in the popliteal fossa, large amplitude M responses were recorded in gastroenemius at a time when only low amplitude H-reflexes were recorded from soleus. These result not only reinforce the importance of casreful selection of the stimulation site but go further and suggest a definite need for the simultaneous monitoring of gastrocnemius and soleus responses to avoid contamination of the soleus H-reflex or its excitability cycle by the unwanted and unsuspected gastrocnemius M response. Conventional practice of recording an H-relfex and its excitability cycle from a single pair of bipolar electrodes located over soleus is susceptible to confounding from the unwanted M response by alterations in the initial mechanical conditions and by the various afferent discharges evoked by a preceding contraction in gastrocnemius.

Animals↗
PubMed1980

QRS mapping in the evaluation of acute anterior myocardial infarction.

In 42 patients with acute anterior myocardial infarction (AMI), we studied the course of Q-wave development and R-wave reduction during the first 48 hours after the onset of chest pain. We used precordial mapping in relation to clinical features, hemodynamic measurements and enzyme release. Q waves developed within 6-14 hours (mean 9 hours) after onset of symptoms. R-wave amplitudes demonstrated nearly a reflected image: They reduced abruptly 5-11 hours (mean 9 hours) after onset of chest pain, coinciding with ST-segment elevation. In 14 patients (group A, 33%) after initial QRS alterations, there were no further changes. Twenty patients (group B, 48%) had a distinct new increase of Q waves (delta sigma Q = 3.0 +/- 2.0 mV/hours) and further R-wave reduction (-delta sigma R = 1.0 +/- 0.6 mV/hour) simultaneous with new severe chest pain and a delayed second increase of enzyme release corresponding with extension of infarction. There were no significant differences between the groups in age, hemodynamics and infarct size calculated from creatine kinase release. Eight patients (group C, 19%) had contradictory findings. Our findings are consistent with previous results indicating that the critical period for intervention is very small except in patients with extension of necrosis.

Adult↗
PubMed1978

[Exercise ECG as a screening test in the early diagnosis of coronary heart disease. Result of a trial study (author's transl)].

1500 men (aged 30-55 years) volunteered in a trial of early recognition of cardiovascular disease. History and clinical as well as biochemical data were obtained and compared with results of exercise ECGs. In the group as a whole abnormal ECGs were obtained in 6.2%, in a subgroup of those without exercise-dependent pain in the thorax, without hypertension and noraml resting ECG it was 3.8%, while in the presence of one or several of these risk factors it was as high as 17.8%. It is concluded that in any screening programme it is not reasonable to perform exercise ECGs in asymptomatic persons without risk factors, because in this group there is likely to be only a small percentage of abnormal findings and the number of false-positive ones is higher than that or correct positive ones. In connection with the known risk factors and risk indicators it would be best to define the risk group and, using selective indications, to go step-by-step from exercise ECG to additional invasive diagnostic measures such as coronary angiography.

Adult↗