Delayed full-thickness skin grafts present an alternative means of treatment to flaps for the closure of deep defects in the lid region. They are indicated wherever there is a risk of recurrence of the causative process which might lead to the loss of the transposed skin.

Eyelid Diseases↗

PubMed1975

[Gross anatomy of the vitreous body (author's transl)].

The anatomical arrangement of vitreous structures in human and animal eyes is described on the basis of coinciding findings of histological examinations, biomicroscopy and the "optical dissection" of unfixed vitreous specimens. The "funnel" of the primary vascular vitreous disappears completely during development. The vitreous cavity is filled with secondary vitreous. Its structure in nearly homogeneous, containing "holes" of low density at all sites where the structure of a normal inner retinal surface is altered. During postnatal development the secondary vitreous forms a new funnel system, consisting of zones of different density and of membranelles (vitreous tracts). Before completion of this funnel system, destruction of the vitreous gel begins and confuses the typical arrangement. Postnatal vitreous structures include: zones of different density: preretinal zone, intermediary zone, retrolental zone; holes (of low density): prepapillary hole, prefoveal hole, prevascular fissures, vitreous tracts (of high density): preretinal tract, median tract, coronary tract, retrolental tract ("hyaloid" tract). The pattern of the "holes" strongly suggests that the framework is formed by retinal elements.

Adult↗

PubMed1975

Autoregulation of renal blood flow in the puppy.

The ability of the immature kidney to autoregulate blood flow was investigated. Renal blood flow was measured by electromagnetic flowmeter. In six puppies, selective blockade of the intrarenal effects of angiotensin II (AII) by [1-sarcosine, 8-alanine]angiotensin II (anti-AII) administered into the renal artery did not change renal blood flow. During selective renal AII blockade, intravenous AII raised perfusion pressure from 76 +/- 2 to 100 +/- 6 mmHg. Renal blood flow increased from 1.59 +/- 0.29 to 1.98 +/- 0.59 ml/g kidney per min, but returned to control levels within 40 s in spite of persistent arterial pressure elevation. In another group of seven puppies, renal blood flow remained constant despite reduction of renal perfusion pressure by aortic constriction to 60 mmHg. In two of these seven puppies intrarenal anti-AII did not abolish autoregulation. Autoregulation of renal blood flow occurs in the puppy and is not influenced by inhibition of angiotensin. The renin-angiotensin system does not appear to be involved in the normal regulation of renal blood flow in the puppy.

Angiotensin II↗

PubMed1975