[Current findings on certain physiopathological aspects of the inappropriate secretion of antidiuretic hormone syndrome].
Hyponatraemia secondary to inappropriate secretion of antidiuretic hormone (SIADH) induces some biological modifications which are relatively characteristic of this condition. The disturbed acid-base equilibrium and the mechanism involved in the high urea and uric acid clearances observed in SIADH are discussed.
Acid-Base Imbalance↗