[The EEG in chronic subdural hematomas in infants].
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Biomedical subjects
Publications and source records attributed to F Rohmer.
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The authors carried out long-term clinical and E.E.G. studies on 100 cases of acute drug poisoning. They made the following conclusions: - in unexplained coma, the E.E.G. can indicate toxic aetiology and may sometimes even suggest which drug is responsible, especially for certain chemical groups (barbiturates, phenothiasines, benzodiazepines); - the E.E.G. picture contributes a valuable indication of the depth of coma; the way in which it evolves affects the prognosis; - the effectiveness of therapy may be judged by monitoring cerebral electrical activity.
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The authors report 9 cases of cortical blindness (7 of vascular origin, 2 due to septic embolism). These were characterised by blindness, loss of the blink reflex in response to threat, and of optokinetic nystagmus, conservation of the pupil reflexes, a confusional state with memory disturbances and anosognosia. The accompanying E.E.G. changes were basically impoverishment of occipital alpha activity, with theta and delta waves mainly in posterior regions on one or both sides; blocking to eye opening was absent, and following of the occiptal background rhythm in response to photic stimulation was rare and partial. One patient developed repetitive slow waves in the left occiptal region, another developed bi-occipital spikes and a third developed bi-occipital paroxymal activity, more predominant on the right. These abnormalities resemble those seen during extraterritorial circulatory insufficiency or air emboli.
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