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Biomedical subjects

F Robinson

Publications and source records attributed to F Robinson.

77 records · Page 5Linked to original sources
PubMedDate not supplied

Spontaneous spinal epidural hematoma.

Two cases of the spontaneous occurrence of spinal epidural hematomas of the high thoracic area are reported. Both acute and subacute presentations of paraplegia are represented. Neither patient had experienced any significant antecedent trauma. No predisposing medical conditions were present. Both patients recovered to independent ambulation following timely operative intervention. The pertinent literature on spinal epidural hematomas is reviewed, and the differential diagnosis of this entity is discussed. The need for prompt diagnosis and surgical treatment to achieve the best neurological outcome is emphasized.

Female↗
PubMedDate not supplied

Femoral neuropathy secondary to anticoagulant therapy.

Two patients with femoral neuropathy resulting from compression by spontaneous hematoma of the iliopsoas muscle while on anticoagulant therapy, were definitively diagnosed preoperatively by CT scan. Early decompression of the femoral nerve, after correcting the coagulopathy, is recommended to assure optimal chances for relief of pain and recovery of neurological function.

Female↗
PubMedDate not supplied

Saccadic dysmetria is similar in patients with a lateral medullary lesion and in monkeys with a lesion of the deep cerebellar nucleus.

Some of the clinical hallmarks of lateral medullary infarction--Wallenberg's syndrome--are saccadic dysmetria, smooth pursuit deficit, and lateropulsion of the body. Similar movement disorders are seen in monkeys after local unilateral injection of GABAergic drugs in the caudal fastigial nucleus of monkeys. These include an ipsilateral saccadic hypermetria and a contralateral saccadic hypometria as well as cogwheel smooth pursuit eye movements toward the contralateral side and an ipsiversive lateropulsion of the body. It was previously suggested that the lateral medullary infarction causes a lesion of climbing fibers to the cerebellum. This lack of climbing fiber input increases the activity of ipsilateral Purkinje cells, which consequently provide too much inhibition of the deep cerebellar nuclei.

Adult↗