Biomedical subjects
F P Brunner
Publications and source records attributed to F P Brunner.
Hypokalaemia, metabolic alkalosis, and hypernatraemia due to "massive" sodium penicillin therapy.
Hypokalaemia and metabolic alkalosis were seen in three patients and additionally hypernatraemia in two patients treated with 100 mega units of sodium penicillin G for subacute bacterial endocarditis. The hypernatraemia was probably due to the administration of insufficient fluid, while urinary potassium loss was an important factor in producing hypokalaemia and metabolic alkalosis after. Penicillin may promote urinary potassium excretion by acting as a non-reabsorbable anion.Potassium depletion during treatment with massive doses of sodium penicillin G may be prevented by concurrently administering potassium-sparing diuretics or by using the potassium salt of penicillin.
[Clinical experiences with the new potassium sparing diuretic amilorid (Moduretic, Colectril, MK-870)].
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[Experiences with peritoneal dialysis in the preparation for kidney transplantation].
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Pitfalls in the use of micropuncture for the localization of diuretic action.
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The mechanism of the urinary concentrating defect in potassium deficient rats.
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Mechanism of glomerulotubular balance. I. Effect of aortic constriction and elevated ureteropelvic pressure on glomerular filtration rate, fractional reabsorption, transit time, and tubular size in the proximal tubule of the rat.
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Mechanism of glomerulotubular balance. II. Regulation of proximal tubular reabsorption by tubular volume, as studied by stopped-flow microperfusion.
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End-stage renal failure due to diabetic nephropathy: data from the EDTA Registry.
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