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Biomedical subjects

F K Bohn

Publications and source records attributed to F K Bohn.

At least 19 recordsLinked to original sources
PubMed1993

[A clinical study of examples of variability and instability of fixed sections of canine EKGs].

Study in ECGs of a clinical collection of 45 dogs (15 dogs without heart disease and without sedation or anesthesia, 15 dogs with acquired and congenital heart disease without sedation or anesthesia, 15 dogs premedicated and anesthetised and all dogs except one without heart disease). Especially studied were the parts which are known in the ECG of the dog as variable and labile (ST-segment, ST-segment-junction and the T-wave). In the group of premedicated and anesthetised dogs a curved and arched ST-segment was more often observed as in comparison to the other two groups. On the other hand the ECG evaluations showed that in all studied groups independent of the status of the heart many similar or equal configurations of the variable and labile parts of the ECG were observed.

Anesthesia↗
PubMed1990

[Anticholinergic medication in the dog before and during anesthesia].

In clinical studies in dogs of all categories of age, which were predicted for surgical purposes under a combination anaesthesia with Fluanisone/Fentanyl/Nitrous oxide/Halothane, investigations after treatment with atropine or glycopyrrolate were performed. In experimental studies investigations about heart-rate and heart work (rate-pressure-product RPP) under different injection anaesthesia-methods (Fluanisone/Fentanyl/Metomidate, Climazolam/Fentanyl, Xylazine/l-Methadone) are performed. In the clinical studies many of the dogs produce elevated heart-rates after anticholinergic premedication. After special indicated treatment of dysrhythmias with glycopyrrolate or atropine in all cases normorhythmia can be achieved. An increase in heart rate during awaking time can be seen in non premedicated as well as in anticholinergic treated animals for a short period of time. In the experimental studies the anticholinergic treatment leads to increased heart rate and/or elevated arterial pressure, which produce an enormous increase in the rate pressure product and oxygen consumption. In conclusion a general anticholinergic premedication can not be recommended. Its use should be special indicated for bradycardia and/or dysrhythmias in the sense of AV-conduction disturbances.

Animals↗
PubMed1990

[Adams-Stokes syndrome and therapy with pacemaker implantation in a dog].

Case report concerning therapeutic pacemaker implantation in a 3 year old West-Highland-White terrier with Adams-Stokes syncopes. EKG's, X-ray-pictures, illustrations of the pulse rate and quality and oxygen measurements with a "Pulsoximeter" before and during surgery are shown. A special place takes the report of selection and control of the anesthesia in this case of a special operation. 17 references and 11 pictures.

Adams-Stokes Syndrome↗
PubMed1981

Evolution of the electrocardiogram in young dogs with congenital heart disease leading to right ventricular hypertrophy.

In 18 pups obtained from a breeding colony established for investigation of hereditary heart disease, ECG's were recorded shortly after birth. The ECG's, as well as clinical examinations, were repeated weekly in pups surviving the first week of life. The QRS modal axis was constructed in the frontal, transverse and sagittal planes. Cardiac catheterization was performed on all pups reaching the age of 8 to 12 weeks. VCG's were taken on selected pups. All dogs were necropsied terminally. In pups with severe pulmonic stenosis a pathological right ventricular hypertrophy was recognized immediately after birth. No evolution to a left ventricular dominance occurred in the serial tracings. Pups with milder grades of right ventricular outflow obstruction showed a normal evolution pattern or some minor deviations from the normal QRS complex evolution. In pups with patent ductus arteriosus (PDA) and left-to-right shunt, there was no difference from the normal ECG evolution within the first 12 weeks of life, except for some increase in amplitude. Two cases of PDA and right-to-left shunt were not different from the "physiologic right ventricular hypertrophy" at birth, but developed a severe right ventricular hypertrophy pattern within the first 12 weeks of life without any clinical signs of a left-to-right shunting within the period.

Animals↗