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Biomedical subjects

F C Koranda

Publications and source records attributed to F C Koranda.

12 recordsLinked to original sources
PubMed1988

The temporalis muscle flap for intraoral reconstruction: technical modifications.

For three years we have used the temporalis muscle flap for reconstruction of intraoral defects. Advantages of this flap are that it is non-hair-bearing, nonbulky, pliable, durable, proximal to intraoral defects, and it results in better articulation and deglutition. We have modified our original technique so that we facilitate transference of the flap by a more anterior transection through the zygomatic arch, we improve the aesthetic reconstruction by use of the transected coronoid process, we produce better function by preventing adhesions and granulation tissue, and we enhance definition of the lingual sulcus by splinting.

Ear↗
PubMed1987

The temporalis muscle flap for intraoral reconstruction.

In this series of seven patients, we used the temporalis muscle flap for reconstruction of tongue and floor-of-mouth defects. For intraoral reconstruction, the temporalis muscle flap has advantages over the standard workhorse flap, the pectoralis major. The temporalis muscle flap is far less bulky, more pliable, non-hair bearing, and in closer proximity to the oral cavity. Use of this muscle does not impair mandibular function. The depression that results after the temporalis muscle has been transferred is minimal. Most of this donor area is covered by hair. The only site where the depression can be significant is at the zygomatic arch, where the tunnel into the mouth is formed after removal of the arch. If the arch is wired back into position, this aesthetic detriment is obviated. In gaining exposure of the zygomatic arch, significant traction can be placed on the soft tissues through which the temporal branch of the facial nerve runs. In one patient, a temporal nerve branch paralysis occurred that required a browpexy; in another patient, there was a transient paresis; and in the others, there was no deficit. The temporalis muscle flap is hardy and durable, and has become our mainstay flap for intraoral reconstruction.

Carcinoma, Squamous Cell↗
PubMed1985

Trapdoor effect in nasolabial flaps. Causes and corrections.

The trapdoor effect is an elevated and bulging deformity of tissue within the semicircular confines of a U-, C-, or V-shaped scar. Various theories to explain this phenomenon are lymphatic and venous obstruction, hypertrophy of the scar, excessive fatty and redundant tissue, beveled wound edges, and contracture of the scar. Our data suggest that scar contracture is the predominant cause of the trapdoor effect in nasolabial flaps. For mild to moderately severe trapdoor deformities, multiple, small Z-plasties about the periphery of the nasolabial flap are indicated. Intralesional triamcinolone acetonide injections may produce a "pharmacologic Z-plasty" effect in some trapdoor deformities. For marked trapdoor deformities, the combination of multiple, small Z-plasties along the semicircular scar and peripheral undermining about the trapdoor defect is the corrective procedure. The trapdoor deformity may be prevented or lessened by peripheral undermining about the recipient site of the flap equal to or greater in area than the recipient site.

Basal Cell Carcinoma↗
PubMed1985

Cheilitis glandularis--a disorder of ductal ectasia.

Cheilitis glandularis is characterized by lip eversion and protrusion with obvious, mucoid-secreting ductal orifices along the mucosal-vermilion boundary. One may see confirmatory microscopic findings of ductal ectasia and metaplasia. Vermilionectomy can be efficacious.

Cheilitis↗
PubMed1981

Antimalarials.

The antimalarials, chloroquine, hydroxychloroquine, and quinacrine, are used primarily for malaria; but they can be beneficial for cutaneous lupus erythematosus (LE), polymorphous light eruption, solar urticaria, and porphyria cutanea tarda. Antimalarials bind to deoxyribonucleic acid (DNA) which prevents DNA and ribonucleic acid (RNA) polymerase reactions and DNA heat inactivation; and they inhibit the LE cell phenomenon, antinuclear antibody reactions, and suppress lymphocyte transformation. By competing with calcium ion, they stabilize membranes and have an anesthetic effect. Their anti-inflammatory potential is due to their inhibition of hydrolytic enzymes, stabilization of lysosomes, interference with prostaglandin synthesis, blocking of chemotaxis, and antagonism of histamine responses. The antimalarials have no sunscreening properties. The most common toxic effects are cutaneous pigmentation, nausea, vomiting, diarrhea, mild ileus, and cycloplegia. There has been a reluctance to use chloroquine and hydroxychloroquine because of the possibility of retinopathy. However, if the "safe" daily dose limit of chloroquine, 2 mg per pound of body weight, and of hydroxychloroquine, 3.5 mg per pound of body weight, is followed, the chance of retinopathy is slight. Quinacrine does not cause retinopathy, but it has more cutaneous side effects than the other two agents.

Antimalarials↗